Mediation Intake Questions: Fill & Download for Free

This Question is really an interesting. I loved to answer this question. You can stake in what you’ve learned and what you need to learn and start doing. Learning is growing.You’re already doing these things in your life but still i want to share with you have a look it once :Smile : I can’t believe you never ever smile once in your life. Regardless how bad things are, but you need to smile in those bad things. smile for some funny things. Make them counts for your future memories.Define Yourself : Give meaning to your life and define yourself. You have to define in every situation in your life. Learn how to shift your beliefs and create your own identity. Make your Identity unique than others.Meditation : Meditation techniques keep you grounded and at peace with yourself. Meditation takes away your body from diaereses. Mediation helps you to remove your whole day stress from your body and makes you feel cool and well.Sleep : Mind your sleep. We used to sleep when we’re tired. We should give a proper rest to our body in order to feel fresh next day. Many doctors says that 6–8 hours sleep is necessary for a human body.Listen to your body and go to sleep.Exercise : This is the main part of our life which we never forgot to take care of our health. Fat body is called the “second home of diseases”. If you have no fat then you are free from all types of diseases. A fit person lives more than his age. “The more you fit the more you live”.Grow your own vegetables : You should grow vegetables in your homes garden which you brought from market because the vegetables you brought from market is not safe as many of farmers give poisonous injection to vegetables which is very dangerous for our health. Grow you own vegetables those are safe for you.If you haven’t start doing all these yet, then do it now like:Read : It expands our view,it opens a gate of knowledge for us. It also helps us to improve our English. By continuous reading of books helps us to gain more knowledge and also make a good habit of reading books.Write : Keep a notebook near your bed and when you wake up in the morning, start free writing immediately. Write anything and everything that comes to you head. You don’t need to be a writer or artist to do this!Respect Time : Know that time is one of the most valuable thing. Plan Your time carefully. Don't waste it!Take Risks : The life is all about moments, whether you took that risk or not. At some point all rich people have taken risks, because if you don't take them, you can't become rich. Take action on your life! Don't be afraid from it!Work Hard : Work hard every day on your ideas, goals and dreams. Don't stop when you're tired, stop when you're done.Try To Avoid These Things In Your Life :Stop Giving Up.Stop Pretending.Stop Smoking or Any Injurious Drug.Stop Drinking Alcohol.Stop Live In a Hurry.Stop Sugar Intake.I hope all these my points helps you.“So, Stop thinking About It Start It Now”

This is quite a tricky one to attempt to answer.In theory, I would look at per capita coconut consumption, estimate the percent of calories it contributes to the average daily intake, and try and find a pattern in the longevity and coconut calories. This however would be simplistic and flawed from the get-go. Privileging the contribution of one food product to expected longevity would be just wrong maths and the wrongest reductionist view of diet and health, an area of knowledge that is at best incomplete, at worst flawed. Longevity expectation projections are complex and sociologically determined and not just mediated by food, exercise, healthcare provision. I would ask questions about how long populations in these countries have been using coconut not least because ancestral nutrition is believed to contribute to longevity expectation [1].But I attempted so here it is.While Indonesia, Philippines, India and Brazil are the top producers, the top consumers are Philippines, EU-27, USA and India. Most producers retain a sizeable chunk for consumption in the home markets. However in India, while data may be averaged over the country, the consumption of coconut varies hugely across the country; also the population base is a multiple of that of the USA and the EU.The longevity expectation at birth respectively, according to Wikipedia as of April the 23rd, 2015, is: Philippines at 71.09, EU 78.82, USA at 78.37, India at 69.89 while Indonesia stands at 70.76, Brazil at 75.35.If we set aside India data for reasons mentioned earlier, it seems Philippines and Indonesia form a kind of cluster while the EU and the USA form another cluster while Brazil sits almost in the middle of them.The analysis of longevity expectation here forth would be about the broader development of these countries than their coconut consumption.In other words, I think it is impossible to tease out the contribution of coconut consumption to longevity expectations without a temporally and otherwise extensive analysis and even then I would not expect anything meaningful to emerge from it given the diverse sources of nutrition persons in these countries use.I will look forward to other answers on this question because unless someone has spent years doing this work already, or is willing to undertake it for this question, it would be pretty hard to address this question.Thanks for the A2A.[1] http://www.ncbi.nlm.nih.gov/pubmed/11368478

This is a complicated question because alcohol has so many different effects.Alcohol in moderation is anti-inflammatory in some ways, while it's pro-inflammatory in others.Red wine also has additional benefits via resveratrol which is anti-inflammatory.One prominent example of moderate alcohol consumption being antiinflammatory comes via a protein called CRP which is a biomarker for inflammation. Here's a link to a study on how moderate drinking reduces CRP levels.[1] Here's another study on that :In a randomised, diet controlled intervention study, Sierksma and colleagues19 studied the effect of four glasses (three glasses for women) of beer or alcohol-free beer (control) with dinner during two successive 3 week periods. The total diet was supplied to subjects who were apparently healthy. Plasma CRP and fibrinogen concentrations decreased by 35% and 12%, respectively (both p < 0.05) after the consumption of beer, as compared to alcohol-free beer consumption.[3]However, it goes well beyond CRP. Many other biomarkers of inflammation are reduced.in 1963, leucocyte motility was shown to be decreased by the presence of ethanol in donor blood ... Several laboratories also studied the effects of ethanol on a variety of inflammatory mediators in animal models. ... most studies indicated an inhibitory effect of low dose ethanol on these mediators, including NF-κβ and various cytokines[3]ethanol treatment prior to bacterial stimulation has an inhibitory effect on inflammatory cytokine production including TNF-α, IL-1β and IL-6 (11,17,18). [5]alcohol inhibits NF-kappaB activation by lipopolysaccharide (LPS) in human monocytes[4]acute alcohol consumption has dual anti-inflammatory effects that involve augmentation of IL-10 and attenuation of monocyte inflammatory responses involving inhibition of NF-κB[7]This reduction in inflammation may be one reason why moderate drinking reduces heart disease rates.[2]Moderate alcohol consumption has been associated with a decreased risk for ischemic heart disease. Increases in high-density lipoprotein-cholesterol may explain approximately 50% of this apparent benefit. Another potential mechanism may involve the effects of alcohol on systemic inflammation.[2]However, alcohol is also pro-inflammatory in some ways, and that may be responsible for some of its effects on the brain. Some of this is mediated via its effects on toll-like receptors.Alcohol consumption can induce brain damage, demyelination, and neuronal death, although the mechanisms are poorly understood. Toll-like receptors are sensors of the innate immune system and their activation induces inflammatory processes. We have reported that ethanol activates and recruits Toll-like receptor (TLR)4 receptors within the lipid rafts of glial cells, triggering the production of inflammatory mediators and causing neuroinflammation.[6]ethanol up-regulates TLR4 and TLR2 expression in microglial cells, inducing the production of inflammatory mediators which triggers reactive oxygen species generation and neuronal apoptosis. [6]Low/moderate ethanol concentrations (10–50 mM, in the range found in the blood of social drinkers and alcoholics) (Adachi et al., 1991) are capable of promoting translocation and clustering of TLR4 and CD14 and the signaling molecules (IRAK, MyD88, ERK) into the lipid rafts (Blanco et al., 2008; Fernandez-Lizarbe et al., 2008). Conversely, high ethanol concentrations or lipid raft-disrupting agents (streptolysin-O or saponin) inhibit ethanol-induced activation of the TLR4 signaling pathway (Blanco et al., 2008; Fernandez-Lizarbe et al., 2008). [8]ethanol at low/moderate concentrations activates the TLR4receptors in astrocytes, triggers NFB activation, and leads to theinduction of an inflammatory response.[8]When consumed to excess, markers of inflammation can increase.Moderate, acute alcohol consumption or equivalent doses of alcohol in vitro had anti-inflammatory effects on monocyte activation via inhibition of pro-inflammatory genes and NF-κB activation, inhibition of TNFα production and augmentation of the anti-inflammatory cytokine, IL-10. In contrast, acute alcohol treatment augmented NF-κB activation and TNFα production and inhibited IL-10 levels in the presence of complex stimulation with combined TLR2 and TLR4 ligands. Prolonged alcohol exposure also resulted in an increase in NF-κB and TNFα production in response to TLR4 stimulation with LPS.[9]It's worth noting that if you drink too much alcohol, your liver simply can't handle it, and your triglyceride levels rise, insulin resistance rises, and your likelihood of becoming obese also rises.So one way to think about it is that moderate alcohol consumption reduces inflammation in some ways that are favorable for your heart and cardiovascular system, while increasing inflammation in a way that's not so great for your brain. Heavy alcohol consumption causes inflammation that is unfavorable for your heart and cardiovascular system.[1] Alcohol Consumption and Plasma Concentration of C-Reactive Protein[2] Alcohol and inflammation: a possib... [Nutr Metab Cardiovasc Dis. 2002][3] http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1768158/[4] Acute alcohol inhibits the induction of... [Alcohol Clin Exp Res. 2002][5] Inhibition of lipopolysaccharide-mediated NFκB activation by ethanol in human monocytes[6] Ethanol induces TLR4/TLR2 association, triggerin... [J Neurochem. 2013][7] Moderate Alcohol Intake in Humans Attenuates Monocyte Inflammatory Responses: Inhibition of Nuclear Regulatory Factor Kappa B and Induction of Interleukin 10 - Mandrekar - 2006 - Alcoholism: Clinical and Experimental Research - Wiley Online Library[8] http://www.jneurosci.org/content/30/24/8285.full.pdf[9] Page on Nih