Pediatric Symptom Checklist: Fill & Download for Free

You might find it helpful to print out a common screening tool for depression and show her how you scored on it. The test will explain common symptoms of depression, so you don't have to figure out how to explain them.Some widely used, well-respected tools (available free online) are:Beck Depression Inventory (probably where I would start if you need help putting words to how you feel)PHQ-9PHQ-9 for AdolescentsPediatric Symptom Checklist - Youth ReportKeep in mind these tools are NOT enough to make a definitive diagnosis - only to suggest high risk or a need for further screening.But, having the questions written out for you, describing the most common symptoms of depression, may be very helpful.You could also print out some symptom checklists and show them what symptoms you're having.For example, here's a nice online tool that gives you a free report with your results at the end: Could it be Depression?Regarding "BPD"…If you meant Borderline Personality Disorder, try:The MSI-BPD (http://www.ottawadepressionalgorithm.ca/download/81_2y10LlGhwqBLL1mkWdDTyn7XecYzOx)The PDQ-4+ (https://www.reginfo.gov/public/do/DownloadDocument?objectID=6088601)If you meant bipolar disorder (BPAD), try:The Mood Disorder Questionnaire (https://www.integration.samhsa.gov/images/res/MDQ.pdf)The Bipolar Spectrum Diagnostic Scale (https://www.psychcongress.com/saundras-corner/scales-screeners/bipolar-disorder/bipolar-spectrum-diagnostic-scale-bsds)Alternately…Google symptom checklists and/or symptom journals for depression and BPD. Print them out and keep a symptom diaryIf you have a smartphone, there are plenty of free symptom diary appsRead personal accounts people have written about their depression from websites like Live Through This (The Survivors) or To Write Love On Her Arms. When you find one that describes how you feel, or tells part of your story, print it out and show it to them.I’m hoping you get to feeling better soon :)

No. We are less than one percent of the population, and less than one percent of our numbers are in prison. The same amount that is the case for neurotypicals. The idea of the criminal psychopath is born out of confirmation bias, not reality.Being identified as psychopathic by the PCL-R, the checklist that they use, is a joke. It identifies antisocial behavior, not psychopathy. It is due to this that there is an absurd belief about psychopathy.Wow, I am reading this study and it is dreck.Psychopathy is astonishingly common as mental disorders go. It is twice as common as schizophrenia, anorexia, bipolar disorder, and paranoia, and roughly as common as bulimia, panic disorder, obsessive-compulsive personality disorder, and narcissism.Ummm…. noSchizophrenia-1%Statistical Prevalence of Schizophrenia. Schizophrenia is not a terribly common disease but it can be a serious and chronic one. Worldwide about 1 percent of the population is diagnosed with schizophrenia, and approximately 1.2% of Americans (3.2 million) have the disorder.Aug 7, 2009Anorexia-3%The National Association of Anorexia Nervosa and Associated Disorders states that approximately eight million people in the U.S. have anorexia nervosa, bulimia, and related eating disorders. Eight million people represents about three percent (3%) of the total population.ANRED: Eating Disorders StatisticsBipolar-2.6%Bipolar Myth No. 1: Bipolar disorder is a rare condition. Not so, according to statistics and research. In a given year, bipolar disorder affects about 5.7 million American adults, or about 2.6% of the U.S. population 18 and older, according to the National Institute of Mental Health.Aug 1, 20088 Bipolar Myths: Symptoms, Mania, Diagnosis, Statistics, and MoreParanoia-2.3%-4.4%People with paranoid personality disorder are generally characterized by ... and occurs somewhere between 2.3 and 4.4 percent in the general population.Paranoid Personality Disorder Symptoms | Psych CentralBulemia-3%The National Association of Anorexia Nervosa and Associated Disorders states that approximately eight million people in the U.S. have anorexia nervosa, bulimia, and related eating disorders. Eight million people represents about three percent (3%) of the total population.ANRED: Eating Disorders StatisticsPanic Disorder 1–2%It's estimated that almost 1 to 2 percent of the general population will suffer with panic disorder in any given year, and approximately 5 percent of the population will experience panic disorder at some point in their lives.Panic Disorder | NewHarbinger.comOCD- 2.3%Approximately 2.3% of the population between ages 18- 54 suffers from OCD, which out ranks mental disorders such as: schizophrenia, bipolar disorder, or panic disorder. In the U.S., approximately 3.3 million people have OCD, of which you have 0.3 to 1% of pediatric population and 2% of adult population.UOCD: Facts& Statistics on OCD.Narcissistic Personality disorder- 5.9–10%According to the largest study ever conducted on personality disorders (PD) by the U.S. National Institutes of Health (NIH), 5.9% of the U.S. population has BPD (Grant et al. 2008) and 6.2% has NPD (Stinson et al. 2008). As some people fit both diagnoses, about 10 percent of the U.S. population has BPD and/or NPD.How common are borderline and narcissistic personality disorders?How they came to the conclusion that something that occurs in .75%-1% of the population is much more prevalent is a mystery to me, but they are ridiculously incorrect.Another reason to trust a Hariot as far as you can throw them. They will skew the data to suite their narrative. It’s too bad that among those that think Hare actually knew what he was doing, not a single one has a shred of critical thinking capability. The very basis of their assumptions are the PCL-R. It’s probably one of the worst “tools” that has ever been dreamed up. There’s a reason the Hare will sue anyone that challenges it. He knows it can’t stand on it’s merits, so he has to shut down any critique that people have of it. Both he and his acolytes are pathetic.https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4059069/

This answer briefly summarizes some overarching inferences,Autism - Wikipedia / Autism spectrum - Wikipedia (Autism Spectrum Disorders, ASD) rates started greatly increasing in some countries such as the US and UK since the 1980s even as doctors little understood these conditions and offered little of value to increasingly anxious parents desperately seeking definitive answers. Thus, in such an Autism causation vacuum, Andrew Wakefield - Wikipedia et al's 1998 Lancet report (1), the first to offer an explanation for the 'autism epidemic', became a convenient crutch for many frustrated parents who felt either ignored or condescended to by the medical establishment.However, in ~20 years, there's surprisingly scant scientific evidence to support the contention that 'vaccines cause autism'. Surprising because 20 years is a long enough period to be able to bolster the argument with solid data sets.Even taken at face value, many risk factors about Autism/ASD simply cannot be explained by a 'vaccines cause autism' notion. The more facts it can explain about a given phenomenon, the stronger a given hypothesis. That is just not the case with the 'vaccines cause autism' notion, which is simply inherently scientifically weak.On a topic so controversial as a potential vaccine(s)-Autism link, it may be best to start by scrutinizing the original data that got this particular idea started. In 1998, Andrew Wakefield and 12 co-authors published a Lancet article on 12 children, claiming they had identified in them evidence of a novel syndrome they called Autistic enterocolitis - Wikipedia (1).To digress just a bit at first, it's somewhat surprising that there isn't yet an agreed-upon consensus on the etiquette regarding scientific papers that have been retracted (2, 3). Specifically, should they continue to be cited in the literature or not? For example, this Wakefield et al paper continues being cited, 85 times already over six months in 2017 according to Google Scholar.This answer however requires not just citing this paper but also looking at what it actually says since it subsequently served as the launchpad for a purported vaccine(s)-Autism link. While 8 of these 12 children (67%) had received the MMR vaccine by the time of their symptom onset, the authors concluded (see below from 1, emphasis mine),'We identify associated gastrointestinal disease and developmental regression in a group of previously normal children, which was generally associated in time with possible environmental triggers...We did not prove an association between measles, mumps, and rubella vaccine and the syndrome described...If there is a causal link between measles, mumps, and rubella vaccine and this syndrome, a rising incidence might be anticipated after the introduction of this vaccine in the UK in 1988. Published evidence is inadequate to show whether there is a change in incidence22 or a link with measles, mumps, and rubella vaccine.23'Since these authors did suggest 'a rising incidence [of their newly coined syndrome] might be anticipated after the introduction of this [MMR] vaccine in the UK in 1988', if we give them the benefit of the doubt and assume their autistic enterocolitis concords to some extent with Autism, what do epidemiological data show so far? In a nutshell, nothing that supports their supposition. On the contrary, such studies haven't found a link between vaccines and Autism.A 1999 study of children in North Thames, London, found rising cases of ASD since 1979 without a sharp increase after MMR was introduced in 1988 (4).A 2001 British study found that while Autism rates in 2 to 5 year olds had increased from 8 boys per 10000 to 29, a 3.6-fold increase, from 1988 to 1993, rates of MMR vaccination had remained stable across these birth cohorts, meaning it wasn't possible to attribute the Autism rate increase to the MMR vaccine (5).Thus an examination of the original paper that jump-started the vaccines-Autism controversy finds it did not even make such an assertion and that subsequent studies found no evidence of such a link either. OTOH, one detailed review after another has since found that the MMR vaccineIs safe (6, 7).Is unlinked to Autism (8).The furore, notoriety and controversy about a link between vaccines and Autism begins with this one study of a mere 12 children, only 8 of whom had received the MMR vaccine by the time of their symptom onset, and it turns out the study didn't even make that claim. Also more accurately, the paper explores not a link between vaccines and Autism in general but rather one specifically between the MMR vaccine and autistic enterocolitis, a syndrome that isn’t listed in medical textbooks.So, how did a link between vaccines and Autism even get made? Turns out to have been a subsequent interpretation (3), perhaps helped along by an immediate press conference when this paper was published followed by copious contemporaneous sensationalist front-page coverage by several British newspapers (9) of a kind that suggests (3) many couldn't even be bothered to read what was actually in the paper.Subsequent uncovering of undisclosed conflicts of interest behind Wakefield's study followed by predictable establishment backlash against him then cast him in the potent 'martyr' mode, which further solidified and enhanced his reputation among parents desperately seeking definitive answers to their children's Autism/ASD diagnosis, and who also felt Wakefield took them seriously while feeling the medical establishment didn't (9).How Autism's Causation Vacuum was Fertile Soil for Wakefield's Vaccine-Autism Supposition to take RootOn the face of it, it seems astounding that one small study on 12 patients should have had such an outsize impact. And yet, maybe not so surprising from a sociological perspective. At the time the Wakefield et al paper came out, Autism/ASD rates had already been spiking for several years with no satisfactory explanation from the medical establishment. Perhaps unwittingly, this state of affairs helped stoke and sustain this particular controversy.Autism diagnosis remains the purview of behavioral scientists who base the diagnosis on a highly subjective checklist, not an impartial, objective, quantitative diagnostic test.Even as they tweaked and improved their diagnostic toolkit, which in turn led to increasing rates of diagnosis, doctors had no clear answer for why steadily increasing numbers of children were being diagnosed with Autism from the 1980s, especially in the US and UK.Still little understood, neither reliable objective diagnosis nor specific treatment, let alone cure, yet existed for Autism/ASD, a situation little changed in the years since.With increasing numbers of parents desperately seeking answers to their children's predicament, a causation vacuum concerning Autism was precisely calamitous and in hindsight, the Wakefield paper appears to have arrived at just the right moment to fill it with something that no one had proposed thus far, a 'medical explanation for the autism epidemic' (see below from 9, emphasis mine).'However, the fact that there was no other reported or known reason for the ‘epidemic’ did not exactly help matters. Whatever their overall validity, vaccine hypotheses did plug a gaping hole in scientific knowledge about this condition that everyone thought had been measured so precisely and accurately with a wealth of new measurement tools and scales. How could it be that no one actually knew why autism was increasing?...Wakefield’s work was so popular because it promised so much. It promised to fully explain the autism epidemic, thus it was particularly ironic that epidemiological sciences never supported his claims.’Autism/ASD having historically been and tending to remain the purview of behavioral scientists may, in the grand scheme of things, turn out to have been a major stumbling block that stymied accelerated understanding of these conditions.Ironically, by highlighting gastrointestinal issues in autistic children, Wakefield may have done Autism/ASD research a huge service. After all, ~20 years on, the gut microbiota-brain link is so much better appreciated now and indeed gut Dysbiosis - Wikipedia is today well-recognized as a cardinal feature in substantial numbers of Autism/ASD patients (10, 11).There was and is an urgent need for a more multi-disciplinary approach for both research and diagnosis in the Autism/ASD field. Gastroenterologists, immunologists, microbiologists, geneticists and other specialists would only help not impede better understanding of these conditions by helping develop more scientifically robust diagnostic approaches and helping tailor more targeted therapies.Even in 2017, such cross-disciplinary research on Autism/ASD is sorely lacking. A simple literature search is a clear indication of this. My search for 'Autism' in both Nature Reviews Immunology and Nature Reviews Microbiology together turned up a total of only 24 articles, 2001-2017 (12), only 19 in Nature Reviews Gastroenterology and Hepatology, though through 2006-2017, which suggests the gut-microbiota-brain axis is becoming a bigger focus of research (13), while the same search in Nature Reviews Neuroscience turned up almost 10X higher articles (219), 2001-2017 (14). For context, the Nature Reviews series are typically considered among the most influential science review journals for various subjects.History also suggests the Wakefield idea fills the Autism/ASD causation vacuum rather like a square peg in a round hole. After all, it is inherently scientifically weak since there are so many Autism/ASD risk factors that effects of vaccines, adverse or otherwise, simply cannot explain.So many Autism/ASD Risk Factors that Vaccines can't explainHow could vaccines possibly explainWhy Autism/ASD is more common in boys than girls, ranging from ~4:1 in the 1990s (15) to ~9:1 by the 2010s (16, 17, 18)? If vaccines 'cause' autism, a person's gender shouldn't matter.Why Autism/ASD rates are so much higher in monozygotic (identical) (70-90% concordance) compared to dizygotic (fraternal) (0-30% concordance) twins (19, 20, 21)?Found in disparate populations such as in the UK (22) as well as in Scandinavia (23).Monozygotic twin concordance for autism is a long-standing feature, being observed right from the 1970s in pioneering studies by Michael Rutter - Wikipedia (24).Autism thus has an unmistakably strong genetic component (22), something that could not be explained by environmental factors alone such as effects of vaccine(s), adverse or otherwise.If vaccines 'cause' autism, a person's genetic background shouldn't matter.Autism/ASD connection with maternal and child antibiotic use reported in several studies (25, 26, 27)? This alludes to a different environmental trigger, namely, changes in gut microbiota composition.Consistently identified Autism risk factors such as exposure to traffic-related air pollutants, increased parental age, maternal obesity, diabetes and folic acid deficiency, prenatal viral infection, C-section, preterm birth, low birth weight, limited or absent breastfeeding, abnormal melatonin synthesis, hyperbilirubinemia, zinc deficiency, and maternal immigrant status (28, 29, 30, 31, 32, 33)? These factors and vaccines are simply unconnected.Autism/ASD are clearly multi-factorial, with both genetic and environmental factors intersecting in as-yet undeciphered ways, and since rates started to increase dramatically since the 1980s, clearly some environmental factor(s) are key. However, those factors still remain stubbornly unclear. Rather than vaccines, however, multiple studies since at least 2004 have consistently reported altered gut microbiota composition in ASD subjects (10, 11). Whether that's cause or effect still remains to be determined.CodaBasing anti-vaccine sentiment on a purported vaccines-autism link is reckless and dangerous since it inflicts real cost in the form of needless deaths from vaccine preventable diseases. Consider measles where the vaccine is historically one of the safest on record. In June 2017, a six year old Italian leukemia patient died from measles complications after reportedly catching it from his older brother, whom his parents had decided not to vaccinate (34), the latest in a measles 'tragedy' that has so far taken 35 lives across Europe (35).Bibliography1. Wakefield, Andrew J., et al. "RETRACTED: Ileal-lymphoid-nodular hyperplasia, non-specific colitis, and pervasive developmental disorder in children." (1998): 637-641. http://www.thelancet.com/pdfs/journals/lancet/PIIS0140-6736(97)11096-0.pdf2. da Silva, Jaime A. Teixeira, and Judit Dobránszki. "Highly cited retracted papers." Scientometrics 110.3 (2017): 1653-1661.3. Collins, Harry M., Luis Reyes‐Galindo, and Paul Ginsparg. "A note concerning primary source knowledge." Journal of the Association for Information Science and Technology 68.5 (2017): 1105-1110. https://arxiv.org/ftp/arxiv/papers/1605/1605.07228.pdf4. Taylor, Brent, et al. "Autism and measles, mumps, and rubella vaccine: no epidemiological evidence for a causal association." The Lancet 353.9169 (1999): 2026-2029. https://www.researchgate.net/profile/Brent_Taylor3/publication/12921734_Autism_and_Measles_Mumps_and_Rubella_Vaccine_No_Epidemiological_Evidence_for_a_Causal_Association/links/02e7e51d45c2c59b90000000.pdf5. Kaye, James A., Maria del Mar Melero-Montes, and Hershel Jick. "Mumps, measles, and rubella vaccine and the incidence of autism recorded by general practitioners: a time trend analysis." Bmj 322.7284 (2001): 460-463. https://pdfs.semanticscholar.org/4657/f7bf358ca0d1c5aa341ed33a63df84e52da7.pdf6. Halsey, Neal A., and Susan L. Hyman. "Measles-mumps-rubella vaccine and autistic spectrum disorder: report from the New Challenges in Childhood Immunizations Conference convened in Oak Brook, Illinois, June 12–13, 2000." Pediatrics 107.5 (2001): e84-e84. http://pediatrics.aappublications.org/content/pediatrics/107/5/e84.full.pdf7. Demicheli, Vittorio, et al. "Vaccines for measles, mumps and rubella in children." Cochrane Database Syst Rev 4.4 (2005). https://www.researchgate.net/profile/Carlo_Di_Pietrantonj/publication/221834605_Vaccines_for_measles_mumps_and_rubella_in_children/links/00463517a5aa79a22b000000/Vaccines-for-measles-mumps-and-rubella-in-children.pdf8. Stratton, Kathleen, et al. "Immunization safety review: measles-mumps-rubella vaccine and autism." (2001). https://www.ncbi.nlm.nih.gov/books/NBK223376/pdf/Bookshelf_NBK223376.pdf9. Evans, Bonnie. The metamorphosis of autism. Manchester University Press, 2017. https://www.ncbi.nlm.nih.gov/books/NBK436841/pdf/Bookshelf_NBK436841.pdf10. Mayer, Emeran A., David Padua, and Kirsten Tillisch. "Altered brain‐gut axis in autism: Comorbidity or causative mechanisms?." Bioessays 36.10 (2014): 933-939.11. Hsiao, Elaine Y. "Gastrointestinal issues in autism spectrum disorder." Harvard review of psychiatry 22.2 (2014): 104-111. https://pdfs.semanticscholar.org/ce91/0afdf1c3da4cb6dc814170733a0204611180.pdf12. nature.com search13. nature.com search14. nature.com search15. Baron-Cohen, Simon, and Jessica Hammer. "Is autism an extreme form of the" male brain"?." Advances in Infancy research 11 (1997): 193-218. https://pdfs.semanticscholar.org/ad8c/4b5c9e91059d95e98649c94c7569c492a3f2.pdf16. Whiteley, Paul, et al. "Gender ratios in autism, Asperger syndrome and autism spectrum disorder." Autism Insights 2 (2010): 17. https://www.researchgate.net/profile/Paul_Whiteley/publication/51018620_Gender_Ratios_in_Autism_Asperger_Syndrome_and_Autism_Spectrum_Disorder/links/0ecba90a0ad20b9df3097710.pdf17. Ruzich, Emily, et al. "Sex and STEM occupation predict autism-spectrum quotient (AQ) scores in half a million people." PloS one 10.10 (2015): e0141229. Sex and STEM Occupation Predict Autism-Spectrum Quotient (AQ) Scores in Half a Million People18. Baron-Cohen, Simon, et al. "Elevated fetal steroidogenic activity in autism." Molecular psychiatry 20.3 (2015): 369. https://www.nature.com/mp/journal/v20/n3/pdf/mp201448a.pdf19. Muhle, Rebecca, Stephanie V. Trentacoste, and Isabelle Rapin. "The genetics of autism." Pediatrics 113.5 (2004): e472-e486. https://www.researchgate.net/profile/Isabelle_Rapin/publication/8583430_The_genetics_of_autism/links/00463521b8de571cdd000000/The-genetics-of-autism.pdf20. Rosenberg, Rebecca E., et al. "Characteristics and concordance of autism spectrum disorders among 277 twin pairs." Archives of pediatrics & adolescent medicine 163.10 (2009): 907-914. https://www.researchgate.net/profile/Rebecca_Rosenberg2/publication/26871866_Characteristics_and_Concordance_of_Autism_Spectrum_Disorders_Among_277_Twin_Pairs/links/561f019a08aec7945a271a75.pdf21. Hallmayer, Joachim, et al. "Genetic heritability and shared environmental factors among twin pairs with autism." Archives of general psychiatry 68.11 (2011): 1095-1102. https://pdfs.semanticscholar.org/3c04/5560f824473172027c89eaeefa46260afe55.pdf22. Bailey, Anthony, et al. "Autism as a strongly genetic disorder: evidence from a British twin study." Psychological medicine 25.1 (1995): 63-77. https://www.researchgate.net/profile/Emily_Simonoff2/publication/15407569_Autism_as_a_Strongly_Genetic_Disorder_Evidence_from_a_British_Twin_Study/links/591d4f3c45851540595c8e43/Autism-as-a-Strongly-Genetic-Disorder-Evidence-from-a-British-Twin-Study.pdf23. Steffenburg, Suzanne, et al. "A twin study of autism in Denmark, Finland, Iceland, Norway and Sweden." Journal of Child Psychology and Psychiatry 30.3 (1989): 405-416.24. Folstein, Susan, and Michael Rutter. "A Twin Study of Individuals with Infantile Autism." Autism. Springer US, 1978. 219-241.25. Konstantareas, M. Mary, and Soula Homatidis. "Brief report: Ear infections in autistic and normal children." Journal of autism and developmental disorders 17.4 (1987): 585-594.26. Niehus, Rebecca, and Catherine Lord. "Early medical history of children with autism spectrum disorders." Journal of Developmental & Behavioral Pediatrics 27.2 (2006): S120-S127.27. Adams, James B., et al. "Mercury, lead, and zinc in baby teeth of children with autism versus controls." Journal of Toxicology and Environmental Health, Part A 70.12 (2007): 1046-1051.28. Landrigan, Philip J. "What causes autism? Exploring the environmental contribution." Current opinion in pediatrics 22.2 (2010): 219-225. http://www.autism-society.org/wp-content/uploads/2014/04/Autism-and-Environment.pdf29. Rossignol, Daniel A., and Richard E. Frye. "A review of research trends in physiological abnormalities in autism spectrum disorders: immune dysregulation, inflammation, oxidative stress, mitochondrial dysfunction and environmental toxicant exposures." Molecular psychiatry 17.4 (2012): 389. https://pdfs.semanticscholar.org/552b/c0709407afc962369a322a41d2ae605a8b16.pdf30. Grabrucker, Andreas M. "Environmental factors in autism." Frontiers in Psychiatry 3 (2012). https://www.researchgate.net/profile/Andreas_Grabrucker/publication/235368604_Environmental_Factors_in_Autism/links/0c96052f1f836e4fc0000000.pdf31. Rossignol, D. A., S. J. Genuis, and R. E. Frye. "Environmental toxicants and autism spectrum disorders: a systematic review." Translational psychiatry 4.2 (2014): e360. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3944636/pdf/tp20144a.pdf32. Ornoy, A., L. Weinstein-Fudim, and Z. Ergaz. "Prenatal factors associated with autism spectrum disorder (ASD)." Reproductive Toxicology 56 (2015): 155-169. https://www.researchgate.net/profile/Asher_Ornoy/publication/277408501_Prenatal_factors_associated_with_Autism_Spectrum_Disorder_ASD/links/55756d7d08aeb6d8c01959b7/Prenatal-factors-associated-with-Autism-Spectrum-Disorder-ASD.pdf33. Ng, Michelle, et al. "Environmental factors associated with autism spectrum disorder: a scoping review for the years 2003-2013." Chronic Diseases and Injuries in Canada 37.1 (2017). http://www.phac-aspc.gc.ca/publicat/hpcdp-pspmc/37-1/assets/pdf/ar-01-eng.pdf34. Child's death from measles caught from unvaccinated brother reignites debate in Italy35. Measles 'tragedy' kills 35 across Europe - BBC NewsThanks for the R2A, Marcos Santiago.