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yes, you do get a small rise in blood pressure from drinking coffee since drinking it leads to a release of adrenaline. If you want to avoid an elevation in blood pressure you would be better served by cutting out alcohol and even more from losing body mass, if you’re overweightThis is NOT something you want to ignore (especially when it can be treated). If you’re struggling with blood pressure take a look at this: Blood Pressure Exercises VSL cbGive this a watch.Text version:I a a > y 6 8 yyy 5 yyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyyi. A b jbjb{x{x $* yy yy yy . . . . .. . ~ ~ 2 2 2 2 2 2 2 2 Z , I R A 14 . 2 2 2 2 2 14 Z . . 2 2 N Z Z Z 2 “ .2 . 2 Z Z B $ f . . .. 2 Z Z Z . . Z & Da{C T : Z Z c 0 Z a Z a Z Z .~ ~ $ d ~ ~ Caffeine causes high blood pressure right? First, some interesting facts about caffeine… 1 It’s the most widely consumed psychoactive substance in the world an estimated 90% of adults in Western countries consume it daily. 2 Small doses of caffeine help reduce pain and speed up the effects of other pain relievers like aspirin by up to 40%. 3 Green tea, oolong tea and black tea are all the same leaves from the same plant, they’re just processed differently. 4 The “buzz” after the first few sips of coffee or tea is largely psychological though some caffeine is absorbed directly into the blood through the mouth, most of it takes over 45 minutes to metabolize in the stomach and small intestine.5 The world’s number one producer of coffee? Is it Columbia? Wrong, try Vietnam. 6 In a typical adult, most of the effects of caffeine pass within 5 to 8 hours: In women taking contraceptives, effects can last 5 to 10 hours and in pregnant women as much as 11 hours. So, getting back to the question… Is it true that caffeine causes high blood pressure? There’s little doubt that, in the short run, caffeine causes blood pressure spikes in people that don’t drink it regularly.As an example, in research published in the New England Journal of Medicine, non coffee drinking study subjects had average blood pressure increases of 14 systolic and 10 diastolic. Whether or not caffeine causes long term hypertension over time in regular drinkers is less clear: One way to find out is through researching “meta studies.” Meta studies take a large number of clinical trials and then reduce the mass of data into a conclusion. For example, in a study published by the American Heart Association called, “The Effect of Chronic Coffee Drinking on Blood Pressure,” they concluded that the average overall increase was 2.4 systolic and 1.2 diastolic. Just to confuse matters, yet another meta study published in the Journal of Hypertension concluded that the effects of caffeine were different depending on if it was from coffee versus other caffeinated drinks : Their final conclusion was, “Regular caffeine intake increases blood pressure. When ingested through coffee, however, the blood pressure effect of caffeine is small.” These numbers may seem small, but when you’re talking about blood pressure, even very small numbers can make a big difference. In population study research published in the Journal of Biobehavioral Medicine, it was estimated that increases as small as these could be attributed to a 14% increase in death from coronary heart disease and a 20% increase in strokes.Like so much about blood pressure, how caffeine affects you personally is very individual. The only way to know for sure? Take your blood pressure before drinking caffeine and then again 30 and 60 minutes later: If you’re one of the people who can safely drink caffeine without getting a blood pressure spike, it doesn’t mean you have a free ticket to drink all you want: It’s generally recommended to drink no more than 350 milligrams a day: Depending on your preferred strength, this is roughly 2 to 4 cups of coffee; or 3 to 9 cups of tea: So there you have it… Does caffeine increase blood pressure? The answer is: maybe… everyone is different.To learn more about natural ways to control your blood pressure without drugs, visit us at: www. Lower. Blood. 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First, I will address the health repercussions of eating chocolate:Both the sugar and fat (cocoa butter) in chocolate have a deleterious effect on your health [1]. The actual cacaw bean solids (you know them as cocoa powder) are beneficial to the point where, according to the leading journal of the American Heart Association, they can be recommended without hesitation [2]. In particular, it was found that Dutch cocoa (raw - unprocessed) was healthier than the processed (and sugar added) cocoa [3].You may also want to read all the Cocoa Good; Chocolate Bad articles (with journal source citations), as well as the Healthiest Chocolate Fix articles (again, with journal source citations).As to whole grains (real whole grains, not processed food with whole grains in them), they seem to extend our lives due to various interactions, one of which is fiber content [4]. Thus, both chocolate (in the form or unprocessed cocoa) and whole grains are excellent for you.A lifetime hint for you - when asking questions like this, make sure replies are supported with a string of reproducible independent peer-reviewed non-conflicting studies. If the person can't cite a series of sources that meet the aforementioned requirements in their reply, then consider it nothing more than personal opinion, armchair science, conjecture and personal hypothesis. Your health is much too valuable to leave to someone's opinion or interpretation, when the actual peer-reviewed facts are readily available.Source Citations:[1] F. Visioli, H. Bernaert, R. Corti, C. Ferri, S. Heptinstall, E. Molinari, A. Poli, M. Serani, H. J. Smit, J. A. Vinson, F. Violi, and R. Paoletti. Chocolate, lifestyle, and health. Crit Rev Food Sci Nutr, 49(4):299-312, 2009.[2] Z. Faridi, V. Y. Njike, S. Dutta, A. Ali, and D. L. Katz. Acute dark chocolate and cocoa ingestion and endothelial function: A randomized controlled crossover trial. American journal of clinical nutrition, 88(1):58, 2008.[3] R. Corti, A. J. Flammer, N. K. Hollenberg, and T. F. Luscher. Cocoa and cardiovascular health. Circulation, 119(10):1433-1441, 2009.[4] Tiwari U, Cummins E. Meta-analysis of the effect of β-glucan intake on blood cholesterol and glucose levels. Nutrition. 2011 Oct;27(10):1008-16.[4] Sun Q, Spiegelman D, van Dam RM, Holmes MD, Malik VS, Willett WC, Hu FB. White rice, brown rice, and risk of type 2 diabetes in US men and women. Arch Intern Med. 2010 Jun 14;170(11):961-9.[4] Law MR, Morris JK, Wald NJ. Use of blood pressure lowering drugs in the prevention of cardiovascular disease: meta-analysis of 147 randomised trials in the context of expectations from prospective epidemiological studies. BMJ. 2009 May 19;338:b1665.[4] Tighe P, Duthie G, Vaughan N, Brittenden J, Simpson WG, Duthie S, Mutch W, Wahle K, Horgan G, Thies F. Effect of increased consumption of whole-grain foods on blood pressure and other cardiovascular risk markers in healthy middle-aged persons: a randomized controlled trial. Am J Clin Nutr. 2010 Oct;92(4):733-40.

By high blood sugar, I will assume you’re referring to hyperglycemia, which is a major health concern, as it can affect people with both type 1 and type 2 diabetes. There are two kinds of hyperglycemia:Fasting hyperglycemia, measured as a blood sugar level exceeding 130 mg/dL after not eating or drinking for at least 8 hours.Postprandial hyperglycemia, measured as a blood sugar level exceeding 180 mg/dL two hours after eating.People without diabetes rarely have blood sugar levels exceeding 140 mg/dL after a meal, unless it’s unusually large. If you have or are at risk for type 2 diabetes, high blood sugar can eventually lead to compromising your body’s ability to process sugar (hyperglycemic hyperosmolar nonketotic syndrome - or HHNS). This, in turn, will increase your risk of atherosclerosis by 100% (that means your risk doubles).It would take at least several book chapters to cover the interactions that lead to this, so I will summarize by giving you some independent peer-reviewed clinical studies published in acknowledged scholarly journals that address each subtle point of the issue, and leave it to you to piece it all together.What causes diabetes:W J Evans. Oxygen-carrying proteins in meat and risk of diabetes mellitus. JAMA Intern Med. 2013 Jul 22;173(14):1335-6. doi: 10.1001/jamainternmed.2013.7399.M Cnop. Fatty acids and glucolipotoxicity in the pathogenesis of Type 2 diabetes. Biochem Soc Trans. 2008 Jun;36(Pt 3):348-52. doi: 10.1042/BST0360348.R Taylor. Banting Memorial lecture 2012: reversing the twin cycles of type 2 diabetes. Diabet Med. 2013 Mar;30(3):267-75. doi: 10.1111/dme.12039.A K Leamy, R A Egnatchik, J D Young. Molecular mechanisms and the role of saturated fatty acids in the progression of non-alcoholic fatty liver disease. Prog Lipid Res. 2013 Jan;52(1):165-74. doi: 10.1016/j.plipres.2012.10.004.D Estadella, C M da Penha Oller do Nascimento, L M Oyama, E B Ribeiro, A R Dâmaso, A de Piano. Lipotoxicity: effects of dietary saturated and transfatty acids. Mediators Inflamm. 2013;2013:137579. doi: 10.1155/2013/137579.R Taylor. Pathogenesis of type 2 diabetes: tracing the reverse route from cure to cause. Diabetologia. 2008 Oct;51(10):1781-9. doi: 10.1007/s00125-008-1116-7.D A Cunha, M Igoillo-Esteve, E N Gurzov, C M Germano, N Naamane, I Marhfour, M Fukaya, J M Vanderwinden, C Gysemans, C Mathieu, L Marselli, P Marchetti, H P Harding, D Ron, D L Eizirik, M Cnop. Death protein 5 and p53-upregulated modulator of apoptosis mediate the endoplasmic reticulum stress-mitochondrial dialog triggering lipotoxic rodent and human β-cell apoptosis. Diabetes. 2012 Nov;61(11):2763-75. doi: 10.2337/db12-0123.G Musso, R Gambino, F De Michieli, M Cassader, M Rizzetto, M Durazzo, E Fagà, B Silli, G Pagano. Dietary habits and their relations to insulin resistance and postprandial lipemia in nonalcoholic steatohepatitis. Hepatology. 2003 Apr;37(4):909-16.J Cao, X X Feng, L Yao, B Ning, Z X Yang, D L Fang, W Shen. Saturated free fatty acid sodium palmitate-induced lipoapoptosis by targeting glycogen synthase kinase-3β activation in human liver cells. Dig Dis Sci. 2014 Feb;59(2):346-57. doi: 10.1007/s10620-013-2896-2.C Xiao, A Giacca, A Carpentier, G F Lewis. Differential effects of monounsaturated, polyunsaturated and saturated fat ingestion on glucose-stimulated insulin secretion, sensitivity and clearance in overweight and obese, non-diabetic humans. Diabetologia. 2006 Jun;49(6):1371-9.M Cnop, S J Hughes, M Igoillo-Esteve, M B Hoppa, F Sayyed, L van de Laar, J H Gunter, E J de Koning, G V Walls, D W Gray, P R Johnson, B C Hansen, J F Morris, M Pipeleers-Marichal, I Cnop, A Clark. The long lifespan and low turnover of human islet beta cells estimated by mathematical modelling of lipofuscin accumulation. Diabetologia. 2010 Feb;53(2):321-30. doi: 10.1007/s00125-009-1562-x.M Ricchi, M R Odoardi, L Carulli, C Anzivino, S Ballestri, A Pinetti, L I Fantoni, F Marra, M Bertolotti, S Banni, A Lonardo, N Carulli, P Loria. Differential effect of oleic and palmitic acid on lipid accumulation and apoptosis in cultured hepatocytes. J Gastroenterol Hepatol. 2009 May;24(5):830-40. doi: 10.1111/j.1440-1746.2008.05733.x.C J Nolan, C Z Larter. Lipotoxicity: why do saturated fatty acids cause and monounsaturates protect against it? 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Towards a new system of health: the challenge of Western disease. J Community Health. 1993 18(1):37-47.If you’re just looking for a general overview of all this, without studying the corresponding peer-reviewed papers, I suggest you watch this hour-long summary that covers atherosclerosis as one of the leading causes of death: