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In the early days, before CSS was even on anyone’s drawing boards, I used have sites running Classic ASP under Microsoft IIS. At the time, there was a lot of variety in screen sizes and creating a small size image left a lot of white space on a high-res screen but creating a large size image required a lot of scrolling on a low-res screen. My solution was to query the browser’s data to find the screen size and dynamically adjust font sizes and image sizes to tailor the content to the screen

Thanks for the A2A.TLDR: different strokes for different folks. I personally like Fuji a lot (so much that I switched away from my Nikon full-frame dSLR to get a Fuji ASP-C body). Olympus has a load of interesting features in a relatively small/affordable package if portability, affordability and speed are more important the absolute image quality. Panasonic has some really great video oriented cameras as well.I wouldn’t say “better,” so much as different. Just like there are a lot of different people with different styles… there are a lot of different cameras with different styles. Canon and Nikon are like the Honda and Toyota of the camera world. They are top sellers and have extremely, test tested and honed competence in photographer. That being said… in their effort to appeal to the largest audience, some people might find them to be a bit bland and risk-adverse for fear of hurting their existing sacred cash cows.Sony is a newcomer with a slightly out of the box thinking of what a camera should/could be and so have make cameras that, for better or worse… eschew a bunch of the old conventions of camera design. Sometimes that’s good because some of the current aspects of camera design are somewhat of a relics of the pre-digital age (e.g. having mirrors). They are pushing the envelope in technical specs with high-res cameras (a7R-series), low light video cameras (a7S-series), and high speed/focus cameras (Sony a9). The a7 III is a great balance of those innovations… but it is not cheap, especially combined with the G-master series lenses. However, Sony still have some more affordable/compact lenses like the 28mm F2 or the 35mm F2.8. While not jaw-dropping, image quality is pretty good for the price.Fuji is an interesting combination of old versus new. They bring back some “retro” design elements (aperture rings on lenses) with some new technology (X-Trans Non-Bayer color filter arrays). They also decided to buck the idea that 35mm (aka full frame) is the ideal sensor size… offering both APS-C and Medium Format camera options (wayback… 35mm was considered the smallest film format… with medium and large formatThey provided some interesting range finder style cameras like the Xpro or X100-series. The X100 series is more than just looks… having a built in ND filter and a leaf shutter (something that is rare among cameras).The Fuji X-T3 (disclaimer: I have the X-T2) was chosen by DPreview as the best camera of 2018 (over the Sony A7 III and Nikon Z6). Not that these kinds of lists are really reliable… but the fact that it is even mentioned in the same breadth as Nikon and Sony shows they are worth considering. They might be not be for you… but it can’t help to check them out.

Europeans have pale skin due to genetic mutations as they lack versions of genes called—SLC24A5, SLC45A2, MFSD12, OCA2, HERC2, the MC1R (and others)— which promotes depigmentation and Pheomelanogenesis. Pale skin is very recent in history occurring as recent as only 8000 years ago. How Europeans evolved white skin“An analysis of 50,000-year-old Neanderthal DNA suggests that Neanderthals had mutations in their MC1R. MC1R is a receptor gene that controls the production of melanin, the protein responsible for pigmentation of the hair and skin. Neanderthals had a mutation in this receptor gene which changed an amino acid, making the resulting protein less efficient and likely creating a phenotype of light hair and pale skin. DNA: Genotypes and PhenotypesThe findings, are based on the sequence of a single gene, called MC1R. Humans with a less functional form of the MC1R protein are more likely to be fair skinned — an adaptation that may have helped inhabitants of high latitudes synthesize vitamin D more efficiently in limited sunlight.” Lalueza-Fox, C. et al. Science doi:10.1126/science.1147417 (2007).It is thought and falsely taught that the reason for lighter skin is due to the need to synthesize Vitamin D (which is actually a secosteriod that transforms into a hormone after sun exposure) at a “better” rate in cold environments with less sunlight, thus Eumelanin turned into Pheomelanin.However, medical research shows that Caucasians and Asians have the highest rates of Osteoporosis and low bone density than any other race. (Defining Ethnic and Racial Differences in Osteoporosis and Fragility Fractures)This conscept is incorrect genetically speaking. The ability of Eumelanin phenotypes to synthesize “Vitamin-D” is just as efficient if not more efficient than Pheomelanin phenotypes ability to maintain properly levels of “Vitamin-D” as explained below and here: Vitamin D–Binding Protein and Vitamin D Status of Black Americans and White Americans | NEJMThe binding of MSH (melanocyte stimulating hormone) to the MC1R (Melanocortin 1 receptor) in the melanocytes stimulates cell production and synthesis of Eumelanin pigments. Eumelanin is known for its ability to absorb UV radiation, where as Pheomelanin, is photo-unstable and sensitive to UV radiation causing it to promote carcinogenesis in many cases. According to Blum (1961) in his dissertation entitled, “Does the melanin pigment of human skin have adaptive value?,” the Vitamin D theory failed to account for the fact that, “the spectral distribution of sunlight at the earth's surface is insufficient to explain the distribution of human skin color differences.” (Blum HF. 1961. Does the melanin pigment of human skin have adaptive value? Q Rev Biol 36: 50– 63.).Furthermore, the production of these two melanins are completely different. Eumelanin follows a completely different pathway than Pheomelanin. The binding of MSH to the MC1R activates the receptor and stimulates eumelanin synthesis.As you can see in this diagram the production of Pheomelanin lacks the MC1R. This mutation was one cause of pale skin, amongst others. Pheomelanogenesis begins with the agouti signal protein (ASP). L-cysteine is the chief source of sulfur and is, therefore, essential for the pheomelanin synthesis. Pheomelanin is a Sulfur based chemical involved in the production of L-Cysteine and GSH which differ from Eumelanogenesis which uses Hydro-Carbons. In studies in mice, the ASIP gene is associated with adult-onset obesity, increased tumor susceptibility, and premature infertility. Pheomelanin also gives off a strong Sulfur smell affecting the skin and hair during perspiration, or contact with moisture. (See below)Eumelanogenesis begins with gene interaction from the Melanocyte stimulating hormone (MSH) and the Melanocortin stimulating hormone (MC1R). “Eumelanin also has the ability to transform photon energy into chemical energy through the dissociation of water molecule, a role performed supposedly only by chlorophyll in plants, seriously questions the sacrosanct role of glucose and thereby mitochondria as the primary source of energy and power for the cells.” https://www.ncbi.nlm.nih.gov/m/pubmed/25645910/ 2H2O ↔ 2H2 + O2 + 4e-“Melanin is thousands of times more efficient to dissociate the water molecule than chlorophyll, it suffices to note that chlorophyll does so irreversible and can only use purple and red light, visible both, for our side, melanin absorbs the entire electromagnetic spectrum, apart from splitting water, also has the amazing ability to re-shape it, which is a unique example in nature.” See: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4125832/Eumelanin is defined as the black and brown pigment and a polymer of dihydroxyindole and carboxylic acid. Pheomelanin is defined as the yellow and red pigment and a benzothiazine derivative. There is no way sunlight could trigger such a monumental change in the genetic code, when in fact it is known that “Vitamin D” is not synthesized “better” by pheomelanotic skin types, whites medically have higher instances of Osteoporosis, (Defining Ethnic and Racial Differences in Osteoporosis and Fragility Fractures) Parkinson’s Disease (Racial Differences in the Diagnosis of Parkinson’s Disease), Multiple Sclerosis (Multiple sclerosis in US minority populations: Clinical practice insights), Infertility, Erectile Dysfunction (Erectile Dysfunction Influenced By Race And Ethnicity), Cardiovascular Disease, and other Vitamin D deficient illnesses. This came on the heels and discovery that “Black People” are not “Vitamin D” deficient. See: Has removal of excess cysteine led to the evolution of pheomelanin?Dr. Ravi Thadhani Et. Al., solved the mystery of blacks being labeled as Vitamin-D Deficient despite not having usual indicators such as low bone density, symptoms of osteoporosis and increased fracture risk since blacks have the best bone health compared to other “races.” The problem lies in the test for 25-hydroxyvitamin D which were based on the levels of whites individuals. Genetics are the cause of some 80 percent of the difference in the “Vitamin D-binding” protein levels. Bone density levels and calcium levels were also higher in blacks compared with white. See: Vitamin D–Binding Protein and Vitamin D Status of Black Americans and White Americans | NEJMFurthermore, Pheomelanin is phototoxic (Phototoxicity is an inflammatory skin reaction induced by skin exposure to a chemical or exposure to sunlight or UV radiation) because it produces reactive oxygen species when exposed to UV radiation. Therefore, the theory that pale skin came from the need to better synthesize Vitmain-D is an incorrect theory. See: MC1R, Eumelanin and Pheomelanin: Their Role in Determining the Susceptibility to Skin CancerIn contrast, there seems to be no physiological or biological benefits in having pheomelanin. Pheomelanin is seen as “an accident of nature” as denoted by Hill HZ, Hill GJ. 2000. UVA, pheomelanin and the carcinogenesis of melanoma. Pigment Cell Res 13: 140– 4. By examples given above, the phototoxicity of Pheomelanin suggests that any photoprotective role of melanins is better suited by Eumelanin.