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Why China has a higher average iq than India?
Well I wrote this answer a long time ago…here’s a new updated answerUpdated Answer: The majority of average IQ samples only reflect the younger age group rather than the total population since China’s fertility rate, infant mortality rate, and low birth-weight percentage is much lower than India's the younger group in China reflects a special small sample so higher average scores are expected.Larger samples including older age groups showed much lower average IQs for China in the 70s-90s.There is no country with a high average IQ in larger samples including every age group (rather than only the younger group) it's just a hoax, myth, or distorted statistics.The real average IQ for every country is around the 70s-90s in larger samples including every age group but nearly every sample only reflects the younger age group under age 20.Every country would have a high average IQ if we count only a special small sample rather than the total population….when the fertility rate, infant mortality rate, and low birth-weight percentage is low the younger group represents a special small sample.A special small sample in India would have a high average IQ but India has the highest young population in the world, an extremely high infant mortality rate, and the 5th highest low birth weight percentage so a high average IQ for the younger group is unlikely.By age 2 humans reach around 75%-80% of their adult brain size. Many factors linked to higher infant mortality rates are also linked to impaired brain development and lower IQ like low birthweight which has been linked to stunted brain growth, smaller brains, and lower IQs. Since so many independent unrelated peer-reviewed studies show the same about low birthweight it's one of the most reliable factors influencing brain development and IQ.China has the very lowest percentage of low birthweight babies in the world at 2% whereas India has the 5th highest at 28% (India - Low-birthweight babies (% of births), China - Low-birthweight babies (% of births)).China also has one of the very lowest fertility rates in the world at 1.6 per 1,000 meaning the younger aged group represents proportionally smaller sample sizes.Many blog sites tried to lie and say there was lots of impoverishment in Vietnam and North Korea but the low birthweight data from UNICEF shows Vietnam and North Korea as among the least impoverished countries in the world at 5.1% low birthweight for Vietnam, 5.7% for North Korea (Vietnam - Low-birthweight babies (% of births), Dem. People's Rep. Korea - Low-birthweight babies (% of births)).Vietnam and North Korea have among the lowest fertility rates and low birthweight percentages in the entire world...they can be considered as among the least impoverished nations based on the low birthweight data even though others tried to lie and claim so.The only countries I can find genetically like East Asian with high fertility rates, infant mortality rates, and low birthweight percentages are Cambodia, Laos, and Myanmar which all ranked at the very bottom of all countries in the world in the popular math IMO contest (has an under age 20 rule).Many countries with higher fertility rates and infant mortality rates did better than Cambodia, Laos, and Myanmar even though many had claimed that East Asians must be genetically talented at mathematics. This matches in exactly to my hypothesis with the fertility rate, infant mortality rate, and low birthweight percentage but not into the genetics hypothesis at all.Since the fertility rate, infant mortality rate, and low birthweight percentage is decreasing worldwide in the future like around the 2040s-2060s many other countries may have higher average IQs than China counting only the younger aged group.So far this hypothesis with the fertility rate, infant mortality rate, and low birthweight percentage remains unrefuted.Anyone can easily refute this hypothesis by pointing out a group that did well on younger age academic tests or samples with a high fertility rate, infant mortality rate, and low birthweight percentage.I think we just live in more of an anti-science type of society where people don't challenge others to falsify or refute a hypothesis…I don’t really see many other answers like that anywhere on Quora.If a hypothesis is not falsifiable then you can't tell if it's true or false which is just the same as saying nothing.Average IQ correlates at around r=-.8 with the infant mortality rate alone but we may be able to gain stronger correlations closer to r=-1.0 using the fertility rate and exact factors linked to impaired brain development and lower IQ like low birthweight.Many countries with lower infant mortality rates still have a higher percentage of low birthweight babies than Vietnam and North Korea.Old answer: Brain birth defects / poor nutrition at early ages as indicated by infant mortality rates.The correlation between infant mortality and IQ is extremely strong r=-.84, nearly perfect among the very strongest correlations found with IQ.Leading causes of infant mortality are brain birth defects (linked to low IQ) , low birth weight (linked to low IQ), and poor nutrition at early ages (linked to low IQ) so the infant mortality rate would give us a great idea about the likelihood of brain birth defects and poor nutrition at early ages which is probably why it correlates so strongly with IQ.There are all types of birth defects, neural-tube defects, and nutritional deficiencies linked to impaired brain development and low IQ (and also directly connected to infant mortality).China's infant mortality rate is like 4 times lower than India's infant mortality rate…this means China has far fewer people with birth defects and poor nutrition linked to low IQ than India does.Let’s compare China’s infant mortality rate with India’s infant mortality rate:This means the worst regions in China in terms of infant mortality rates are better than many of the best regions in India in terms of infant mortality rates!The worst regions in China only have infant mortality rates like 16–22 per 1,000 which is lower than nearly every Indian state’s infant mortality rate.China had a one-child policy since 1979…this may explain why China’s infant mortality rate is so low.If India or any country wants to increase their average IQ they should focus on reducing birth defects and poor nutrition at early ages (nearly the same as reducing the infant mortality rate).Maybe passing a one-child or two-child policy like China did would work effectively to reduce the infant mortality rate. Polls show that the majority of Chinese people support the one-child policy (76%) I’m sure many people in India would also support it or a similar policy. It would cause the population growth and infant mortality rate to go down, and also increase the average IQ of the next generation.The idea that the Chinese would still have high average IQs with poor nutrition at early ages has been thoroughly debunked by many peer-reviewed studies like this study showing the Chinese having IQs less than 70:"In Baihuyaon villagers who were aged 30-35 years (n = 50), who were born during the period of severe iodine deficiency, 72% of villagers had an intelligence-quotient score of less than 70" (Iodine deficiency impairs intellectual and neuromotor development in apparently-normal persons. A study of rural inhabitants of north-central China. )The US in the 1900s had an average IQ of 67–70…the infant mortality rate was extremely high back then. The US introduced iodized salt in the mid-1920s which boosted the IQ and reduced the infant mortality rate dramatically. Right now India’s infant mortality rate is similar to the US’ from the 1940s.World Map of Average IQ:World Map of Infant Mortality Rates:You can see how strong the correlation between infant mortality and IQ is by looking at those two maps..it’s as if average IQ and infant mortality rates are one in the same.Groups genetically close but distant in terms of infant mortality rates have average IQs more distant than groups genetically distant but close together in terms of infant mortality rates.Reducing the infant mortality rate of any group or country should increase the average IQ of the next generation dramatically.Every country should have like an average IQ in the 100s-110s once you eliminate birth defects and poor nutrition at early ages which is like the same as having a low infant mortality rate.We should be able to achieve global IQ equality just by achieving a low infant mortality/birth defect/poor nutrition at early ages rate worldwide.I feel that if countries want to improve the brains of the next generation they should use the education funding towards reducing brain birth defects and poor nutrition at early ages (nearly the same as reducing the infant mortality rate) rather than like on improving the education system, new school supplies, or fancy infrastructure, higher pay for teachers, or other things unrelated to brain development. It’s not that complicated or expensive either.Reducing birth defects, NTDs, low birth weight, and poor nutrition at early ages should be the most effective way of increasing the average IQ of any group. If the infant mortality rate is reduced dramatically the next generation would have average IQs like 1 or more SDs above the previous.If India or any country really wants to dramatically increase the average IQ of the next generation they should concentrate their efforts primarily on improving prenatal care, maternal nutrition before and during pregnancy, reducing birth defects, underweight birth, and poor nutrition at early ages…which is nearly the same as reducing the infant mortality rate.Nearly every study shows that nutrition at early ages has a big effect on brain development and IQ but after around age 5 or so it seems to not matter much…probably because by age 5 humans reach 90% of their adult brain size. Nutrition seems to matter a lot at earlier ages…by age 2 humans reach 80% of their adult brain size.Malnutrition is linked to nearly half of infant deaths (45%) so simply having food and water would dramatically reduce the infant mortality rate. Then there are certain things like folic acid which seems to reduce the likelihood of birth defects significantly. Perhaps India should have the fortification of folic acid, iodine, iron, B12, and other things in commonly eaten foods like many other countries do to reduce the infant mortality rate.I wonder if we could create a super-high IQ society where the average IQ is like 160 or so just by copying certain aspects of the diet that high IQ adults had at early ages (and aspects of their mother’s diet before and during pregnancy). I also noticed that Christopher Langan (one of the few people to have an adult IQ of 190 or higher on multiple tests) ate a lot during his growth spurts and started weightlifting at age 12…studies show that for adults weightlifting boosts brain power…but I wonder what effect it would have at earlier ages prior to the brain being fully developed.But you don’t need a high average IQ for survival or everyone in a country to be a super-genius…I wonder why China still has nearly the same amount of Nobel prizes in science as India even with an infant mortality rate 4 times lower…What if a high average IQ isn’t a good thing for society…what if it’s better to have an average IQ in between too high and too low with just some super-geniuses….or what if even a low average IQ is better overall in the long run…
What are the biological arguments against evolution?
1/ Changing the definition of evolution to mean “all changes over time”. Has no direction and is meaningless to the original theory. Of course the weather changes but it doesn’t evolve into something else. Of course each successive generation changes but it does not evolve, it degrades in every complex creature on the planet.Here is the original theory as stated at the conception.Evolution: "that theory which sees in the history of all things organic and inorganic a development from simplicity to complexity, a gradual advance from a simple or rudimentary condition to one that is more complex and of a higher character." Webster's Encyclopedic Dictionary of the English Language.2/ Without this positive beneficial mutation that does not exist anywhere in evidence, there is no advancement from simple to complex.Please find this mutation. Without it you have no theory of creatures advancing from simple life to complex as the belief that fish eventually evolved into humans.*Positive/beneficial mutation is that it must increase complexity NOT be associated with any disease, not be part of normal immune responses to pathogens and/or any of the following: 1/ add new genes with new functions 2/ increase health and fitness to survive 3/ increase intelligence.This is why from the American National Library of Medicine on all the possible types of mutations and the results. You can clearly see from this video that the Library of Medicine is honest and show what is really going on. There is no “model” no way for any increased complexity to ever have a chance in the real world.Are Humans Getting Better? What is Really Going On? by James ArjunaJames Arjuna's answer to How do people go through evolution?There is ONLY degeneration shown in ALL HUMAN studies of DNA and in all the physical evidence known to mankind.There are well over 20,000,000 (updated nov 2017) peer reviewed papers from all over the world clearly showing that humans are degraded, have never "evolved" and at the present rate of the rise of genetic defects our species will be reduced to stupid short living animals with no technology, no speech, no intelligence. We are about 80 years from that, and then rapid extinction because we live only by artificial life support (vaccines, antibiotic, drugs, surgery, medical means all artificial).Search NCBI databasesThere are NO beneficial mutations ever found in DNA evidence.Evolution's "Beneficial" Mutations by James ArjunaTry this search term and see if you can find any beneficial mutations. I have spent thousands of hours on that and so far not one beneficial mutation has ever been shown to exist. This search of all the data bases in the world sums up with over 2,000,000 (2017) articles and NO beneficial mutations.Human Mutations - PMC - NCBIIn all these peer Science studies we find some estimated 20,000 pseudo genes. Pseudo genes are genes that used to code for human cells. Now they are dead and fading away. Some of them are barely perceivable because of this fading of this coding. They have completely mapped and studied 14000 of them so far. They say that some are redundant, but considering the health conditions of modern humans that makes no sense.Analysis of Human PseudogenesThe GENCODE pseudogene resourceAccording to this study we have lost close to 45% of our coding that made us much stronger and much more healthy, fit and much more intelligent.For those who do not understand math:Original number of coding genes approximately 45,000; Coding genes we have now approximately 25,000. Number of pseudo genes (dead genes) no longer coding for body parts 20,000. 20,000 + 25,000 = 45,000 original genes.45,000 x 45% = 20250 lost genes. Actual precise number is 20,000/45,000 = .444444 or 44.4%.Dr. Gerald Crabtree a renowned geneticists and professor of genetics at Stanford University states that“I would wager that if an average citizen from Athens of 1000 BC were to appear suddenly among us, he or she would be among the brightest and most intellectually alive of our colleagues and companions, with a good memory, a broad range of ideas, and a clear-sighted view of important issues. Furthermore, I would guess that he or she would be among the most emotionally stable of our friends and colleagues. I would also make this wager for the ancient inhabitants of Africa, Asia, India or the Americas, of perhaps 2000–6000 years ago. The basis for my wager comes from new developments in genetics, anthropology, and neurobiology that make a clear prediction that our intellectual and emotional abilities are genetically surprisingly fragile.”Human intelligence is declining according to Stanford geneticistLeading Geneticist: Human Intelligence is Slowly DecliningWe have lost;-Brain Size, and skull size-Jaw Size and lost teeth, the 'wisdom" teeth are remnants of a better stronger larger jaw.-Our larger canine teeth that allowed us to eat more foods.-enzymes in the appendix that gave us the ability to digest harsh fibrous vegetation and get nutrients out of it.-visual acuity.-visual spectrum and color (there are a very few people left with tetrachromatic vision)-night vision.-Our retinas are misshaped and we have blind spots we have to overcome by brain adjustments and glasses. Most degraded mammals have this, by the way.-lost http://intelligencehttp://rt.com/usa/intelligence-stanford-years-fragile-531/-lost tails (coccyx) which show as an atavism, atavism is lost genes-lost olfactory sensors in our nasal passages; Vomeronasalorgan, Can’t smell preditors from a distance or food or water.-less bone density; we break bones easily-bone shape, bent crooked leg bones and much thinner than our ancestors.-hair; We cannot live in the wild without clothing or technology-flexibility in our feet our ancestors could grasp in order to climb, we can’t.-cartilage is weak and breaks easily. The number one injury with athletes ripped out tendons in the knees.-hearing loss and weak. Common for humans to lose hearing frequency early in life.-back strength 80% of humans complain of back pain and many have to have medical aid for it.-knee problems from weak thin knee meniscus and bone joints. Many people have Bone to Bone when this fails and intense suffering.-More and more women have to have C section for birth because the “choice” breeders have skinny hips for some unknown reason. Childbirth is the most dangerous of all the medical practices because the malpractice insurance is up to $300,000 per year; whereas internal medicine is $20,000 per year. There’s more about this on the birth defects article.-we cannot produce Vitamin C and many enzymes used to process foods.-eye protective membrane gone “nictitating membrane”- Along with lost hair we see remnants of ‘goose bumps” which are used to raise hair to increase insulation. You see this in animals when it is cold the hair “fluffs up”Going away fading out:-A muscle that show varying degrees of reduction ;Occipitalis Minor-The palmaris longus muscle appears gone in about 15%-17% of humans going away.-The pyramidalis muscle of the abdomen gone in 20% of humans-in the leg, The plantaris muscle absent in about 10% of humans-Extra nipples or breasts appear rarely in the population- the existing sense of smell organs degraded in most and we find remnants of superior sense of smell in native South Americans, native North Americans, and African peoples who can identify other by smell; lost ability to sense predators or food and water.-human immune system weakened and undirected causing all sorts of problems and the need for artificial means of survival, vaccines, and antibiotics were not needed for 200,000 years, now we can’t live without them.There is no indication at all of any improvements. All the nonsense about how "refined and improved" we are in utter fantasy.What are we going to lose next?This is why the medical industry is stating that modern humans, particularly white Anglo Saxons, white European descent cannot live past 20 years in the wild without technology and medical aid.Keep in mind for 200,000 years (by the assumptive dating methods that have never been proven as correct) humans survived, bred and raised children without any medical at all.And there is no way for use to determine how old someone from the past was. Even 5000 years ago, a 100 year old person could appear to be like a modern 40 year old because we are that much more degraded.In Human DNA studies we have over 247,000 deleterious mutation found so far and no sign of any beneficial mutation have ever been verified other than by speculation from people who believe we evolved and got better at some point in time in the past where human science was "different" than now.This is the “magical time” of the past where humans supposedly evolved. There is no physical evidence of this anywhere on the planet.Search NCBI databasesThere have been found average of over 98000 ERV's (endogenous retrovirus). 247,000 total in all of the human race found so far. These represent infections a the germ line level. Infections at the germ line level cause fetal permanent deleterious mutations. The path of our genetic degradation is mapped clearly in these ERVs which are found in DNA coding for diseases tissues and gene losses, atavism.Colloquium Paper: Therapeutic Vaccines: Realities of Today and Hopes for Tomorrow: Retroelements and the human genome: New perspectives on an old relationEvidence for Hitchhiking of Deleterious Mutations within the Human GenomeSung Chun1, Justin C. Fay1,2*1 Computational and Systems Biology Program, Washington University, St. Louis, Missouri, United States of America, 2 Department of Genetics and Center for Genome Sciences and Systems Biology, Washington University, St. Louis, Missouri, United States of America:"Evidence for Hitchhiking of Deleterious Mutations within the Human Genome"According to this article of 1000 samples of human genomes, there is an average of 1000 deleterious mutations per person. Quote: "Each human is estimated to carry on the order of 1,000 deleterious mutations in their genome."These are FIXED (permanent) in the reproductive process.The governments do not want anyone to be concerned and want you to believe that "big brother and science" Is taking good care of us and don't worry about 200% rise in babies with brain/spinal tumors. or the 1 in 64 babies born with autism (up 650% in 39 years); The medical industry will find a cure. Right???So far not one disease has been cured by science.Home - ACCOThey just continue to rise because of human ignorance.Medical Industry:Without the medical industry humans would already be extinct.We have over 17000 genetic disease and so many pathogens that would wipe us out that humans cultivated out of ignorance, we would not be here without antibiotic, anti-fungal, vaccines. This is obvious from the evidence.They have managed to make vaccines to stop polio and small pox but that is not a cure. The cure would be to have all humans not be affected by them at all with no vaccine (restored immune DNA). And for sure they have no cures for genetic deformity except corrective surgery and deleterious mutations. I don’t think they have enough brain power left to even know how to or what to do without killing more people or making mutant freaks from accidents out of them, in the process. With heart surgery it is "plumbing work" and attaching parts of arteries taken from other parts of the body. Cataract surgery is wonderful but does nothing about the genetic defects; the cause nor the cure. Medical industry has enzyme replacements, hormone replacements lens replacements, and a huge number of medicines that reduce body functions to help with pain etc. Cancer treatments are horrible and the remission rate is not that good. They give statistics on 5 years of life after cancer as "success" . In the meantime the rates of cancer rise at phenomenally rapid increases. The childhood cancer is just to harsh to believe. A disease that has no record of death for children in 1847 and I still have found not one case in 1900 on a death certificate. And now it is the number one disease killer of children. Second is genetic deformity, and third is congenital heart disease and that one is up over 200% in the last 34 years.^ Muller, G. B. (2002) "Vestigial Organs and Structures." in Encyclopedia of Evolution. Mark Pagel, editor in chief, New York: Oxford University Press. pp. 1131–1133.Jump up^ http://news.nationalgeographic.com/news/2009/07/090730-spleen-vestigial-organs.html "Vestigial Organs Not So Useless After All, Studies Find"Jump up^ Wiedersheim, Robert (1893). The Structure of Man: an index to his past history. London: Macmillan and Co.Jump up^ Wiedersheim, Robert Ernst Eduard. The structure of man an index to his past history. Macmillan 1895. May be downloaded from [1]^ Jump up to:a b Wells, H.g. Huxley, J. Wells, G. P. The Science of Life. Pub. Cassell 1931Jump up^ Rosenthal, M. I.: Journal of the American Medical Association, Volume 67, Issues 15-26, 1916. Page 1326Jump up^ W. Colin MacKenzie. A Contribution to the Biology of the Vermiform Appendix. Medical record, Volume 89 Page 342 1916^ Jump up to:a b c d e f Darwin, Charles (1871). The Descent of Man, and Selection in Relation to Sex. John Murray: London.Jump up^ Stevens, C. Edward; Hume, Ian (2004). Comparative Physiology of the Vertebrate Digestive System. Cambridge: Cambridge University Press. ISBN 9780521617147.Jump up^ Peter Robert Cheeke, Ellen S. Dierenfeld, Comparative Animal Nutrition and Metabolism. Publisher: CABI; 2010 ISBN 978-1845936310Jump up^ Useful AppendixJump up^ Randal Bollinger, R.; Barbas, Andrew S.; Bush, Errol L.; Lin, Shu S.; Parker, William (2007). "Biofilms in the large bowel suggest an apparent function of the human vermiform appendix". Journal of Theoretical Biology 249 (4): 826–31. doi:10.1016/j.jtbi.2007.08.032. PMID 17936308.Jump up^ Charles Q. Choi, "The Appendix: Useful and in Fact Promising", Live Science, 2009, Appendix has useful functionJump up^ Saraga-Babić, M; Lehtonen, E; Svajger, A; Wartiovaara, J (1994). "Morphological and immunohistochemical characteristics of axial structures in the transitory human tail".Annals of Anatomy 176 (3): 277–86. PMID 8059973.Jump up^ Fallon, John F.; Simandl, B. Kay (1978). "Evidence of a role for cell death in the disappearance of the embryonic human tail". American Journal of Anatomy 152 (1): 111–29. doi:10.1002/aja.1001520108. PMID 677043.Jump up^ Dao, Anh H.; Netsky, Martin G. (1984). "Human tails and pseudotails". Human Pathology 15 (5): 449–53. doi:10.1016/S0046-8177(84)80079-9. PMID 6373560.Jump up^ Dubrow, Terry J.; Wackym, Phillip Ashley; Lesavoy, Malcolm A. (1988). "Detailing the Human Tail". Annals of Plastic Surgery 20 (4): 340–4. doi:10.1097/00000637-198804000-00009. PMID 3284435.Jump up^ Johnson, Dr. George B. "Evidence for Evolution". (Page 12) Txtwriter Inc. 8 Jun 2006.Jump up^ Rozkovcová, E; Marková, M; Dolejsí, J (1999). "Studies on agenesis of third molars amongst populations of different origin". Sbornik lekarsky 100 (2): 71–84.PMID 11220165.Jump up^ Pereira, T. V.; Salzano, FM; Mostowska, A; Trzeciak, WH; Ruiz-Linares, A; Chies, JA; Saavedra, C; Nagamachi, C et al. (2006). "Natural selection and molecular evolution in primate PAX9 gene, a major determinant of tooth development". Proceedings of the National Academy of Sciences 103 (15): 5676–81. doi:10.1073/pnas.0509562103.PMC 1458632. PMID 16585527.Jump up^ Trotier, D.; Eloit, C; Wassef, M; Talmain, G; Bensimon, JL; Døving, KB; Ferrand, J (2000). "The Vomeronasal Cavity in Adult Humans". Chemical Senses 25 (4): 369–80.doi:10.1093/chemse/25.4.369. PMID 10944499.Jump up^ Kjær, Inger; Hansen, Birgit Fischer (1996). "The human vomeronasal organ: prenatal developmental stages and distribution of luteinizing hormone-releasing hormone".European Journal of Oral Sciences 104 (1): 34–40. doi:10.1111/j.1600-0722.1996.tb00043.x. PMID 8653495.Jump up^ Smith, Timothy D.; Siegel, Michael I.; Bhatnagar, Kunwar P. (2001). "Reappraisal of the vomeronasal system of catarrhine primates: Ontogeny, morphology, functionality, and persisting questions". The Anatomical Record 265 (4): 176–92. doi:10.1002/ar.1152. PMID 11519019.Jump up^ Smith, Timothy D.; Bhatnagar, Kunwar P. (2000). "The human vomeronasal organ. Part II: prenatal development". Journal of Anatomy 197 (3): 421–36. doi:10.1046/j.1469-7580.2000.19730421.x. PMC 1468143. PMID 11117628.Jump up^ Won, J; Mair, EA; Bolger, WE; Conran, RM (2000). "The vomeronasal organ: an objective anatomic analysis of its prevalence". Ear, nose, & throat journal 79 (8): 600–5.PMID 10969469.Jump up^ Johnson, A; Josephson, R; Hawke, M (1985). "Clinical and histological evidence for the presence of the vomeronasal (Jacobson's) organ in adult humans". The Journal of otolaryngology 14 (2): 71–9. PMID 4068105.Jump up^ Foltán, René; Šedý, Jiří (2009). "Behavioral changes of patients after orthognathic surgery develop on the basis of the loss of vomeronasal organ: a hypothesis". Head & Face Medicine 5: 5. doi:10.1186/1746-160X-5-5. PMC 2653472. PMID 19161592.Jump up^ Bhatnagar, Kunwar P.; Smith, Timothy D. (2001). "The human vomeronasal organ. III. Postnatal development from infancy to the ninth decade". Journal of Anatomy 199(Pt 3): 289–302. doi:10.1046/j.1469-7580.2001.19930289.x. PMC 1468331. PMID 11554506.^ Jump up to:a b c Bhatnagar, Kunwar P.; Kennedy, Ray C.; Baron, Georg; Greenberg, Richard A. (1987). "Number of mitral cells and the bulb volume in the aging human olfactory bulb: A quantitative morphological study". The Anatomical Record 218 (1): 73–87. doi:10.1002/ar.1092180112. PMID 3605663.Jump up^ Witt, M; Hummel, T (2006). "Vomeronasal Versus Olfactory Epithelium: Is There a Cellular Basis for Human Vomeronasal Perception?". International Review of Cytology248: 209–59. doi:10.1016/S0074-7696(06)48004-9. PMID 16487792.Jump up^ Wysocki CJ, Preti G (November 2004). "Facts, fallacies, fears, and frustrations with human pheromones". The Anatomical Record. Part a, Discoveries in Molecular, Cellular, and Evolutionary Biology 281 (1): 1201–11. doi:10.1002/ar.a.20125. PMID 15470677.Jump up^ Wyatt, Tristram D. (2003). Pheromones and Animal Behaviour: Communication by Smell and Taste. Cambridge: Cambridge University Press. p. 295. ISBN 0-521-48526-6.Jump up^ Prof. A. Macalister, Annals and Magazine of Natural History, vol. vii., 1871, p. 342.Jump up^ Mr. St. George Mivart, Elementary Anatomy, 1873, p. 396.Jump up^ Owen, R. 1866–1868. Comparative Anatomy and Physiology of Vertebrates. London.Jump up^ Montagna, W.; Machida, H.; Perkins, E.M. (1966). "The skin of primates XXXIII.: The skin of the angwantibo". American Journal of Physical Anthropology 25 (3): 277–290.doi:10.1002/ajpa.1330250307. PMID 5971502.Jump up^ Blank, Hanne (2007). Virgin: The Untouched History. Bloomsbury Publishing. pp. 23. ISBN 1-59691-010-0Jump up^ Blackledge, Catherine (2004). The Story of V. Rutgers University Press. ISBN 0-8135-3455-0. "Hymens, or vaginal closure membranes or vaginal constrictions, as they are often referred to, are found in a number of mammals, including llamas, ..."Jump up^ Macalister A (1875). "Observations on muscular anomalies in the human anatomy. Third series with a catalogue of the principal muscular variations hitherto published".Trans. Roy. Irish Acad Sci 25: 1–130.Jump up^ Guerra, A. B.; Metzinger, SE; Metzinger, RC; Xie, C; Xie, Y; Rigby, PL; Naugle Jr, T (2004). "Variability of the Postauricular Muscle Complex: Analysis of 40 Hemicadaver Dissections". Archives of Facial Plastic Surgery 6 (5): 342–7. doi:10.1001/archfaci.6.5.342. PMID 15381582.Jump up^ Tamatsu, Y; Tsukahara, K; Hotta, M; Shimada, K (2007). "Vestiges of vibrissal capsular muscles exist in the human upper lip". Clinical Anatomy 20 (6): 628–31.doi:10.1002/ca.20497. PMID 17458869.Jump up^ Kapoor, SK; Tiwari, A; Kumar, A; Bhatia, R; Tantuway, V; Kapoor, S (2008). "Clinical relevance of palmaris longus agenesis: common anatomical aberration". Anatomical science international 83 (1): 45–8. doi:10.1111/j.1447-073X.2007.00199.x. PMID 18402087.Jump up^ Sebastin, SJ; Lim, AY; Bee, WH; Wong, TC; Methil, BV (2005). "Does the absence of the palmaris longus affect grip and pinch strength?". Journal of hand surgery (Edinburgh, Scotland) 30 (4): 406–8. doi:10.1016/j.jhsb.2005.03.011. PMID 15935531.Jump up^ Rubinstein, David; Escott, Edward J.; Hendrick, Laura L. (April 1999). "The prevalence and CT appearance of the levator claviculae muscle: a normal variant not to be mistaken for an abnormality". AJNR Am J Neuroradiol (American Society of Neuroradiology) 20 (4): 583–6. PMID 10319965.Jump up^ Loukas, M.; Sullivan, A.; Tubbs, R.S.; Shoja, M.M. (2008). "Levator claviculae: a case report and review of the literature". Folia Morphol. 67 (4): 307–310.Jump up^ Lovering, RM; Anderson, LD (2008). "Architecture and fiber type of the pyramidalis muscle". Anatomical science international 83 (4): 294–7. doi:10.1111/j.1447-073X.2007.00226.x. PMID 19159363.Jump up^ Darwin, Charles. (1872) The Expression of the Emotions in Man and Animals John Murray, London.[page needed]Jump up^ Peter Gray (2007). Psychology (fifth ed.). Worth Publishers. p. 66. ISBN 0-7167-0617-2.Jump up^ Behavior Development in Infants (via Google Books) by Evelyn Dewey, citing a study "Reflexes and other motor activities in newborn infants: a report of 125 cases as a preliminary study of infant behavior" published in the Bull. Neurol. Inst. New York, 1932, Vol. 2, pp. 1–56.Jump up^ Jerry Coyne (2009). Why Evolution is True. Penguin Group. pp. 85–86. ISBN 9780670020539.Jump up^ Anthony Stevens (1982). Archetype: A Natural History of the Self. Routledge & Kegan Paul. p. 87. ISBN 0-7100-0980-1.Jump up^ Ohta, Y; Nishikimi, M (1999). "Random nucleotide substitutions in primate nonfunctional gene for L-gulono-gamma-lactone oxidase, the missing enzyme in L-ascorbic acid biosynthesis". Biochimica et Biophysica Acta 1472 (1–2): 408–11. doi:10.1016/S0304-4165(99)00123-3. PMID 10572964.Jump up^ Nishikimi M, Fukuyama R, Minoshima S, Shimizu N, Yagi K (May 6, 1994). "Cloning and chromosomal mapping of the human nonfunctional gene for L-gulono-gamma-lactone oxidase, the enzyme for L-ascorbic acid biosynthesis missing in man". J. Biol. Chem. 269 (18): 13685–8. PMID 8175804.
Has Darwin's theory of evolution been completely refuted? If so, why?
1/ Changing the definition of evolution to mean “all changes over time”. Has no direction and is meaningless to the original theory. Of course the weather changes but it doesn’t evolve into something else. Of course each successive generation changes but it does not evolve, it degrades in every complex creature on the planet.Here is the original theory as stated at the conception.Evolution: "that theory which sees in the history of all things organic and inorganic a development from simplicity to complexity, a gradual advance from a simple or rudimentary condition to one that is more complex and of a higher character." Webster's Encyclopedic Dictionary of the English Language.2/ Without this positive beneficial mutation that does not exist anywhere in evidence, there is no advancement from simple to complex.Please find this mutation. Without it you have no theory of creatures advancing from simple life to complex as the belief that fish eventually evolved into humans.*Positive/beneficial mutation is that it must increase complexity NOT be associated with any disease, not be part of normal immune responses to pathogens and/or any of the following: 1/ add new genes with new functions 2/ increase health and fitness to survive 3/ increase intelligence.***This is why from the American National Library of Medicine on all the possible types of mutations and the results. You can clearly see from this video that the Library of Medicine is honest and show what is really going on. There is no “model” no way for any increased complexity to ever have a chance in the real world.***Are Humans Getting Better? What is Really Going On? by James ArjunaThere is ONLY degeneration shown in ALL HUMAN studies of DNA and in all the physical evidence known to mankind.There are well over 20,000,000 (updated nov 2017) peer reviewed papers from all over the world clearly showing that humans are degraded, have never "evolved" and at the present rate of the rise of genetic defects our species will be reduced to stupid short living animals with no technology, no speech, no intelligence. We are about 80 years from that, and then rapid extinction because we live only by artificial life support (vaccines, antibiotic, drugs, surgery, medical means all artificial).Search NCBI databases (Search NCBI databases)There are NO beneficial mutations ever found in DNA evidence.Evolution's "Beneficial" Mutations by James Arjuna (Evolution's "Beneficial" Mutations by James Arjuna)Try this search term and see if you can find any beneficial mutations. I have spent thousands of hours on that and so far not one beneficial mutation has ever been shown to exist. This search of all the data bases in the world sums up with over 2,000,000 (2017) articles and NO beneficial mutations.Human Mutations - PMC - NCBI (Human Mutations - PMC - NCBI)In all these peer Science studies we find some estimated 20,000 pseudo genes. Pseudo genes are genes that used to code for human cells. Now they are dead and fading away. Some of them are barely perceivable because of this fading of this coding. They have completely mapped and studied 14000 of them so far. They say that some are redundant, but considering the health conditions of modern humans that makes no sense.Analysis of Human Pseudogenes (Analysis of Human Pseudogenes)The GENCODE pseudogene resource (The GENCODE pseudogene resource)According to this study we have lost close to 45% of our coding that made us much stronger and much more healthy, fit and much more intelligent.For those who do not understand math:Original number of coding genes approximately 45,000; Coding genes we have now approximately 25,000. Number of pseudo genes (dead genes) no longer coding for body parts 20,000. 20,000 + 25,000 = 45,000 original genes.45,000 x 45% = 20250 lost genes. Actual precise number is 20,000/45,000 = .444444 or 44.4%.Dr. Gerald Crabtree a renowned geneticists and professor of genetics at Stanford University states that“I would wager that if an average citizen from Athens of 1000 BC were to appear suddenly among us, he or she would be among the brightest and most intellectually alive of our colleagues and companions, with a good memory, a broad range of ideas, and a clear-sighted view of important issues. Furthermore, I would guess that he or she would be among the most emotionally stable of our friends and colleagues. I would also make this wager for the ancient inhabitants of Africa, Asia, India or the Americas, of perhaps 2000–6000 years ago. The basis for my wager comes from new developments in genetics, anthropology, and neurobiology that make a clear prediction that our intellectual and emotional abilities are genetically surprisingly fragile.”Human intelligence is declining according to Stanford geneticist (Human intelligence is declining according to Stanford geneticist)Leading Geneticist: Human Intelligence is Slowly Declining (Leading Geneticist: Human Intelligence is Slowly Declining)We have lost;-Brain Size, and skull size-Jaw Size and lost teeth, the 'wisdom" teeth are remnants of a better stronger larger jaw.-Our larger canine teeth that allowed us to eat more foods.-enzymes in the appendix that gave us the ability to digest harsh fibrous vegetation and get nutrients out of it.-visual acuity.-visual spectrum and color (there are a very few people left with tetrachromatic vision)-night vision.-Our retinas are misshaped and we have blind spots we have to overcome by brain adjustments and glasses. Most degraded mammals have this, by the way.-lost http://intelligencehttp://rt.com/usa/intelligence-stanford-years-fragile-531/-lost tails (coccyx) which show as an atavism, atavism is lost genes-lost olfactory sensors in our nasal passages; Vomeronasalorgan, Can’t smell preditors from a distance or food or water.-less bone density; we break bones easily-bone shape, bent crooked leg bones and much thinner than our ancestors.-hair; We cannot live in the wild without clothing or technology-flexibility in our feet our ancestors could grasp in order to climb, we can’t.-cartilage is weak and breaks easily. The number one injury with athletes ripped out tendons in the knees.-hearing loss and weak. Common for humans to lose hearing frequency early in life.-back strength 80% of humans complain of back pain and many have to have medical aid for it.-knee problems from weak thin knee meniscus and bone joints. Many people have Bone to Bone when this fails and intense suffering.-More and more women have to have C section for birth because the “choice” breeders have skinny hips for some unknown reason. Childbirth is the most dangerous of all the medical practices because the malpractice insurance is up to $300,000 per year; whereas internal medicine is $20,000 per year. There’s more about this on the birth defects article.-we cannot produce Vitamin C and many enzymes used to process foods.-eye protective membrane gone “nictitating membrane”- Along with lost hair we see remnants of ‘goose bumps” which are used to raise hair to increase insulation. You see this in animals when it is cold the hair “fluffs up”Going away fading out:-A muscle that show varying degrees of reduction ;Occipitalis Minor-The palmaris longus muscle appears gone in about 15%-17% of humans going away.-The pyramidalis muscle of the abdomen gone in 20% of humans-in the leg, The plantaris muscle absent in about 10% of humans-Extra nipples or breasts appear rarely in the population- the existing sense of smell organs degraded in most and we find remnants of superior sense of smell in native South Americans, native North Americans, and African peoples who can identify other by smell; lost ability to sense predators or food and water.-human immune system weakened and undirected causing all sorts of problems and the need for artificial means of survival, vaccines, and antibiotics were not needed for 200,000 years, now we can’t live without them.There is no indication at all of any improvements. All the nonsense about how "refined and improved" we are in utter fantasy.What are we going to lose next?This is why the medical industry is stating that modern humans, particularly white Anglo Saxons, white European descent cannot live past 20 years in the wild without technology and medical aid.Keep in mind for 200,000 years (by the assumptive dating methods that have never been proven as correct) humans survived, bred and raised children without any medical at all.And there is no way for use to determine how old someone from the past was. Even 5000 years ago, a 100 year old person could appear to be like a modern 40 year old because we are that much more degraded.In Human DNA studies we have over 247,000 deleterious mutation found so far and no sign of any beneficial mutation have ever been verified other than by speculation from people who believe we evolved and got better at some point in time in the past where human science was "different" than now.This is the “magical time” of the past where humans supposedly evolved. There is no physical evidence of this anywhere on the planet.Search NCBI databases (Search NCBI databases)There have been found average of over 98000 ERV's (endogenous retrovirus). 247,000 total in all of the human race found so far. These represent infections a the germ line level. Infections at the germ line level cause fetal permanent deleterious mutations. The path of our genetic degradation is mapped clearly in these ERVs which are found in DNA coding for diseases tissues and gene losses, atavism.Colloquium Paper: Therapeutic Vaccines: Realities of Today and Hopes for Tomorrow: Retroelements and the human genome: New perspectives on an old relation (Colloquium Paper: Therapeutic Vaccines: Realities of Today and Hopes for Tomorrow: Retroelements and the human genome: New perspectives on an old relation)Evidence for Hitchhiking of Deleterious Mutations within the Human Genome (Evidence for Hitchhiking of Deleterious Mutations within the Human Genome)Sung Chun1, Justin C. Fay1,2*1 Computational and Systems Biology Program, Washington University, St. Louis, Missouri, United States of America, 2 Department of Genetics and Center for Genome Sciences and Systems Biology, Washington University, St. Louis, Missouri, United States of America:"Evidence for Hitchhiking of Deleterious Mutations within the Human Genome"According to this article of 1000 samples of human genomes, there is an average of 1000 deleterious mutations per person. Quote: "Each human is estimated to carry on the order of 1,000 deleterious mutations in their genome."These are FIXED (permanent) in the reproductive process.The governments do not want anyone to be concerned and want you to believe that "big brother and science" Is taking good care of us and don't worry about 200% rise in babies with brain/spinal tumors. or the 1 in 64 babies born with autism (up 650% in 39 years); The medical industry will find a cure. Right???So far not one disease has been cured by science.Home - ACCO (Home - ACCO)They just continue to rise because of human ignorance.Medical Industry:Without the medical industry humans would already be extinct.We have over 17000 genetic disease and so many pathogens that would wipe us out that humans cultivated out of ignorance, we would not be here without antibiotic, anti-fungal, vaccines. This is obvious from the evidence.They have managed to make vaccines to stop polio and small pox but that is not a cure. The cure would be to have all humans not be affected by them at all with no vaccine (restored immune DNA). And for sure they have no cures for genetic deformity except corrective surgery and deleterious mutations. I don’t think they have enough brain power left to even know how to or what to do without killing more people or making mutant freaks from accidents out of them, in the process. With heart surgery it is "plumbing work" and attaching parts of arteries taken from other parts of the body. Cataract surgery is wonderful but does nothing about the genetic defects; the cause nor the cure. Medical industry has enzyme replacements, hormone replacements lens replacements, and a huge number of medicines that reduce body functions to help with pain etc. Cancer treatments are horrible and the remission rate is not that good. They give statistics on 5 years of life after cancer as "success" . In the meantime the rates of cancer rise at phenomenally rapid increases. The childhood cancer is just to harsh to believe. A disease that has no record of death for children in 1847 and I still have found not one case in 1900 on a death certificate. And now it is the number one disease killer of children. Second is genetic deformity, and third is congenital heart disease and that one is up over 200% in the last 34 years.^ Muller, G. B. (2002) "Vestigial Organs and Structures." in Encyclopedia of Evolution. Mark Pagel, editor in chief, New York: Oxford University Press. pp. 1131–1133.Jump up^ http://news.nationalgeographic.com/news/2009/07/090730-spleen-vestigial-organs.html (http://news.nationalgeographic.com/news/2009/07/090730-spleen-vestigial-organs.html) "Vestigial Organs Not So Useless After All, Studies Find"Jump up^ Wiedersheim, Robert (1893). The Structure of Man: an index to his past history. London: Macmillan and Co.Jump up^ Wiedersheim, Robert Ernst Eduard. The structure of man an index to his past history. Macmillan 1895. May be downloaded from [1]^ Jump up to:a b Wells, H.g. Huxley, J. Wells, G. P. The Science of Life. Pub. Cassell 1931Jump up^ Rosenthal, M. I.: Journal of the American Medical Association, Volume 67, Issues 15-26, 1916. Page 1326Jump up^ W. Colin MacKenzie. A Contribution to the Biology of the Vermiform Appendix. Medical record, Volume 89 Page 342 1916^ Jump up to:a b c d e f Darwin, Charles (1871). The Descent of Man, and Selection in Relation to Sex. John Murray: London.Jump up^ Stevens, C. Edward; Hume, Ian (2004). Comparative Physiology of the Vertebrate Digestive System. Cambridge: Cambridge University Press. ISBN 9780521617147.Jump up^ Peter Robert Cheeke, Ellen S. Dierenfeld, Comparative Animal Nutrition and Metabolism. Publisher: CABI; 2010 ISBN 978-1845936310Jump up^ Useful AppendixJump up^ Randal Bollinger, R.; Barbas, Andrew S.; Bush, Errol L.; Lin, Shu S.; Parker, William (2007). "Biofilms in the large bowel suggest an apparent function of the human vermiform appendix". Journal of Theoretical Biology 249 (4): 826–31. doi:10.1016/j.jtbi.2007.08.032. PMID 17936308.Jump up^ Charles Q. Choi, "The Appendix: Useful and in Fact Promising", Live Science, 2009, Appendix has useful functionJump up^ Saraga-Babić, M; Lehtonen, E; Svajger, A; Wartiovaara, J (1994). "Morphological and immunohistochemical characteristics of axial structures in the transitory human tail".Annals of Anatomy 176 (3): 277–86. PMID 8059973.Jump up^ Fallon, John F.; Simandl, B. Kay (1978). "Evidence of a role for cell death in the disappearance of the embryonic human tail". American Journal of Anatomy 152 (1): 111–29. doi:10.1002/aja.1001520108. PMID 677043.Jump up^ Dao, Anh H.; Netsky, Martin G. (1984). "Human tails and pseudotails". Human Pathology 15 (5): 449–53. doi:10.1016/S0046-8177(84)80079-9. PMID 6373560.Jump up^ Dubrow, Terry J.; Wackym, Phillip Ashley; Lesavoy, Malcolm A. (1988). "Detailing the Human Tail". Annals of Plastic Surgery 20 (4): 340–4. doi:10.1097/00000637-198804000-00009. PMID 3284435.Jump up^ Johnson, Dr. George B. "Evidence for Evolution". (Page 12) Txtwriter Inc. 8 Jun 2006.Jump up^ Rozkovcová, E; Marková, M; Dolejsí, J (1999). "Studies on agenesis of third molars amongst populations of different origin". Sbornik lekarsky 100 (2): 71–84.PMID 11220165.Jump up^ Pereira, T. V.; Salzano, FM; Mostowska, A; Trzeciak, WH; Ruiz-Linares, A; Chies, JA; Saavedra, C; Nagamachi, C et al. (2006). "Natural selection and molecular evolution in primate PAX9 gene, a major determinant of tooth development". Proceedings of the National Academy of Sciences 103 (15): 5676–81. doi:10.1073/pnas.0509562103.PMC 1458632. PMID 16585527.Jump up^ Trotier, D.; Eloit, C; Wassef, M; Talmain, G; Bensimon, JL; Døving, KB; Ferrand, J (2000). "The Vomeronasal Cavity in Adult Humans". Chemical Senses 25 (4): 369–80.doi:10.1093/chemse/25.4.369. PMID 10944499.Jump up^ Kjær, Inger; Hansen, Birgit Fischer (1996). "The human vomeronasal organ: prenatal developmental stages and distribution of luteinizing hormone-releasing hormone".European Journal of Oral Sciences 104 (1): 34–40. doi:10.1111/j.1600-0722.1996.tb00043.x. PMID 8653495.Jump up^ Smith, Timothy D.; Siegel, Michael I.; Bhatnagar, Kunwar P. (2001). "Reappraisal of the vomeronasal system of catarrhine primates: Ontogeny, morphology, functionality, and persisting questions". The Anatomical Record 265 (4): 176–92. doi:10.1002/ar.1152. PMID 11519019.Jump up^ Smith, Timothy D.; Bhatnagar, Kunwar P. (2000). "The human vomeronasal organ. Part II: prenatal development". Journal of Anatomy 197 (3): 421–36. doi:10.1046/j.1469-7580.2000.19730421.x. PMC 1468143. PMID 11117628.Jump up^ Won, J; Mair, EA; Bolger, WE; Conran, RM (2000). "The vomeronasal organ: an objective anatomic analysis of its prevalence". Ear, nose, & throat journal 79 (8): 600–5.PMID 10969469.Jump up^ Johnson, A; Josephson, R; Hawke, M (1985). "Clinical and histological evidence for the presence of the vomeronasal (Jacobson's) organ in adult humans". The Journal of otolaryngology 14 (2): 71–9. PMID 4068105.Jump up^ Foltán, René; Šedý, Jiří (2009). "Behavioral changes of patients after orthognathic surgery develop on the basis of the loss of vomeronasal organ: a hypothesis". Head & Face Medicine 5: 5. doi:10.1186/1746-160X-5-5. PMC 2653472. PMID 19161592.Jump up^ Bhatnagar, Kunwar P.; Smith, Timothy D. (2001). "The human vomeronasal organ. III. Postnatal development from infancy to the ninth decade". Journal of Anatomy 199(Pt 3): 289–302. doi:10.1046/j.1469-7580.2001.19930289.x. PMC 1468331. PMID 11554506.^ Jump up to:a b c Bhatnagar, Kunwar P.; Kennedy, Ray C.; Baron, Georg; Greenberg, Richard A. (1987). "Number of mitral cells and the bulb volume in the aging human olfactory bulb: A quantitative morphological study". The Anatomical Record 218 (1): 73–87. doi:10.1002/ar.1092180112. PMID 3605663.Jump up^ Witt, M; Hummel, T (2006). "Vomeronasal Versus Olfactory Epithelium: Is There a Cellular Basis for Human Vomeronasal Perception?". International Review of Cytology248: 209–59. doi:10.1016/S0074-7696(06)48004-9. PMID 16487792.Jump up^ Wysocki CJ, Preti G (November 2004). "Facts, fallacies, fears, and frustrations with human pheromones". The Anatomical Record. Part a, Discoveries in Molecular, Cellular, and Evolutionary Biology 281 (1): 1201–11. doi:10.1002/ar.a.20125. PMID 15470677.Jump up^ Wyatt, Tristram D. (2003). Pheromones and Animal Behaviour: Communication by Smell and Taste. Cambridge: Cambridge University Press. p. 295. ISBN 0-521-48526-6.Jump up^ Prof. A. Macalister, Annals and Magazine of Natural History, vol. vii., 1871, p. 342.Jump up^ Mr. St. George Mivart, Elementary Anatomy, 1873, p. 396.Jump up^ Owen, R. 1866–1868. Comparative Anatomy and Physiology of Vertebrates. London.Jump up^ Montagna, W.; Machida, H.; Perkins, E.M. (1966). "The skin of primates XXXIII.: The skin of the angwantibo". American Journal of Physical Anthropology 25 (3): 277–290.doi:10.1002/ajpa.1330250307. PMID 5971502.Jump up^ Blank, Hanne (2007). Virgin: The Untouched History. Bloomsbury Publishing. pp. 23. ISBN 1-59691-010-0Jump up^ Blackledge, Catherine (2004). The Story of V. Rutgers University Press. ISBN 0-8135-3455-0. "Hymens, or vaginal closure membranes or vaginal constrictions, as they are often referred to, are found in a number of mammals, including llamas, ..."Jump up^ Macalister A (1875). "Observations on muscular anomalies in the human anatomy. Third series with a catalogue of the principal muscular variations hitherto published".Trans. Roy. Irish Acad Sci 25: 1–130.Jump up^ Guerra, A. B.; Metzinger, SE; Metzinger, RC; Xie, C; Xie, Y; Rigby, PL; Naugle Jr, T (2004). "Variability of the Postauricular Muscle Complex: Analysis of 40 Hemicadaver Dissections". Archives of Facial Plastic Surgery 6 (5): 342–7. doi:10.1001/archfaci.6.5.342. PMID 15381582.Jump up^ Tamatsu, Y; Tsukahara, K; Hotta, M; Shimada, K (2007). "Vestiges of vibrissal capsular muscles exist in the human upper lip". Clinical Anatomy 20 (6): 628–31.doi:10.1002/ca.20497. PMID 17458869.Jump up^ Kapoor, SK; Tiwari, A; Kumar, A; Bhatia, R; Tantuway, V; Kapoor, S (2008). "Clinical relevance of palmaris longus agenesis: common anatomical aberration". Anatomical science international 83 (1): 45–8. doi:10.1111/j.1447-073X.2007.00199.x. PMID 18402087.Jump up^ Sebastin, SJ; Lim, AY; Bee, WH; Wong, TC; Methil, BV (2005). "Does the absence of the palmaris longus affect grip and pinch strength?". Journal of hand surgery (Edinburgh, Scotland) 30 (4): 406–8. doi:10.1016/j.jhsb.2005.03.011. PMID 15935531.Jump up^ Rubinstein, David; Escott, Edward J.; Hendrick, Laura L. (April 1999). "The prevalence and CT appearance of the levator claviculae muscle: a normal variant not to be mistaken for an abnormality". AJNR Am J Neuroradiol (American Society of Neuroradiology) 20 (4): 583–6. PMID 10319965.Jump up^ Loukas, M.; Sullivan, A.; Tubbs, R.S.; Shoja, M.M. (2008). "Levator claviculae: a case report and review of the literature". Folia Morphol. 67 (4): 307–310.Jump up^ Lovering, RM; Anderson, LD (2008). "Architecture and fiber type of the pyramidalis muscle". Anatomical science international 83 (4): 294–7. doi:10.1111/j.1447-073X.2007.00226.x. PMID 19159363.Jump up^ Darwin, Charles. (1872) The Expression of the Emotions in Man and Animals John Murray, London.[page needed]Jump up^ Peter Gray (2007). Psychology (fifth ed.). Worth Publishers. p. 66. ISBN 0-7167-0617-2.Jump up^ Behavior Development in Infants (via Google Books) by Evelyn Dewey, citing a study "Reflexes and other motor activities in newborn infants: a report of 125 cases as a preliminary study of infant behavior" published in the Bull. Neurol. Inst. New York, 1932, Vol. 2, pp. 1–56.Jump up^ Jerry Coyne (2009). Why Evolution is True. Penguin Group. pp. 85–86. ISBN 9780670020539.Jump up^ Anthony Stevens (1982). Archetype: A Natural History of the Self. Routledge & Kegan Paul. p. 87. ISBN 0-7100-0980-1.Jump up^ Ohta, Y; Nishikimi, M (1999). "Random nucleotide substitutions in primate nonfunctional gene for L-gulono-gamma-lactone oxidase, the missing enzyme in L-ascorbic acid biosynthesis". Biochimica et Biophysica Acta 1472 (1–2): 408–11. doi:10.1016/S0304-4165(99)00123-3. PMID 10572964.Jump up^ Nishikimi M, Fukuyama R, Minoshima S, Shimizu N, Yagi K (May 6, 1994). "Cloning and chromosomal mapping of the human nonfunctional gene for L-gulono-gamma-lactone oxidase, the enzyme for L-ascorbic acid biosynthesis missing in man". J. Biol. Chem. 269 (18): 13685–8. PMID 8175804.
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