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The so-called Spanish-Flu actually started in the United States and possibly Europe (United Kingdom, Germany and France) in February 1918 and ended in April 2020. Spain declared itself a neutral country during World War-1 and as a result, its government did not censor the news like other nation’s that participated in WW1.The Spanish media reported the countries first cases of the virus during May 1919. Spanish media reports about the virus made their way across the globe and due to confusion it became known as the Spanish-Flu.Despite World War-1 officially ending in 1918, soldiers played a huge role in spreading the virus around the globe. For example, the US President Woodrow Wilson was said to have caught and died from the virus while negotiating the Versailles Treaty during 1919.Moreover, Western soldiers and their allies were sent across the globe to consolidate their victories and to occupy many countries. Many soldiers took their families with them and as a result, this process helped the Spanish-Flu spread across the world.The major reason why the Spanish-Flu ended was because it had infected almost 500 million people and killed between 20-50 million people. Most people that survived infection developed antibodies to protect themselves from reinfection.The Spanish-Flu mutated just like the current Corona-virus (UK, South African & USA mutant variants) and became even more deadlier. The biggest difference between the Spanish Flu and SARS-CoV-2 is the mortality rate (i.e., death rate).The mutated variant of the Spanish-Flu killed the people it infected within hours or days! This is the major reason why the Spanish-Flu ended so quickly.The Spanish flu was Influenza A Subtype H1N1 type virus. Like the coronavirus, the people did not have immunity for the virus. It was highly infectious and spread through respiratory droplets and Aerosol Transmission.What Type/Family Did The 1918 Influenza Virus Come fromUnlike the Coronavirus- SARS-CoV-2 virus, the 1918 Spanish Influenza virus was a Type A Subtype-H1N1 Avian Virus. Hemagglutinin and neuraminidase protein keys helped the 1918 virus enter and infect cells in both animals and people (Gillespie, 2020, University of Minnesota, 2008, & Devitt, 2004).Both viruses were Novel (new) from an animal and mutated to infect people (Birds and Bats). Both viruses have the ability to jump from species and to mutate/evolve and reinvent themselves.The 1981 Spanish Type A Virus originated in birds, mutating to proliferate in human beings. The same virus mutated again during late 1919 to become more deadly and infectious (Gillespie, 2020, University of Minnesota, 2008, University Of Wisconsin-Madison & Devitt, 2004).Mortality Rate Of the 1918 Spanish-FluUnlike SARS-CoV-2, that kills fewer than 2-5% of its victims, the Spanish-Flu was so effective at killing people that it eventually killed itself off! The Spanish-Flu killed about 10–20& of people that it infected.Although people aged younger than 5 years old and older than 65 years of age were considered to be at risk from the 1918 influenza virus, people within the 20 to 40 age range were considered to be at high risk from infection and death from the virus. Thus far, we have not developed adequate information about how the current coronavirus impacts children and adolescents. (Gillespie, 2020, University Of Wisconsin-Madison & Devitt, 2004).The 1918 Influenzas virus also infected mice and caused infection of the lungs & severe haemorrhaging just like it did to people. Many people reported experiencing many weird and non-flu-like virus symptoms.Many people died from bacterial super-infections due to the lack of antibiotics (especially during 1918). After losing more than 20 million people, the world population developed a collective immunity against the virus in 1920.Measures Used To Eradicate The 1918 Spanish Flu VirusAuthorities locked down areas where there was a virus cluster, made people wear masks, implemented social distancing, infection control measures, and other measures we have taken during the 2020 Coronavirus Pandemic. The above-mentioned measures were relatively successful at protecting people from the 1918 virus and to eradicate it.Most people infected with the virus developed a life-long natural immunity to the virus. Survivors of the 1918 virus still have antibodies in their blood for the 1918 virus!It is disheartening, to say the least, that people do not develop lasting immunity and antibodies for the current coronavirus. In fact, those with a low viral load during infection do not develop antibodies or their antibodies only protect them for 3–6 months.People used masks, social distancing, borders were closed and other infection control strategies helped contain the virus. In fact, fear drove people to lock themselves indoors and to avoid social contact.Finally, back in 1918 people did not travel as much and as easily as we do today. The fact that we did not have millions of people travelling internationally for tourism and business really helped in ending the Spanish-Flu pandemic.References:Gillespie, C. (2020). Explore Health- How Are the Spanish Flu and COVID-19 Alike? Here's What Doctors Say. Viewed at: How Are the Spanish Flu and COVID-19 Alike? Here's What Doctors Say | Health.com, viewed on 18022021.University of Minnesota Centre for infectious disease Research and Policy (2008). Researchers find long-lived immunity to 1918 pandemic virus. Viewed at: Researchers find long-lived immunity to 1918 pandemic virus | CIDRAP (umn.edu), viewed on 19/02/2021).University of Wisconsin-Madison & Devitt. (2004). Gene from 1918 virus proves key to virulent influenza. Viewed at: Gene from 1918 virus proves key to virulent influenza (wisc.edu), viewed on 18/02/2021).

I go to grad school at University of Wisconsin Madison, which is ranked #11 in US News Rankings: Computer Science. Apologies if the following reads like a marketing blurb, but hey, that's what the OP asked for.The systems and networking groups here are pretty awesome. We regularly publish in top conferences, win best paper and other research awards, get a ton of grants, and all the other good stuff you'd find in a top research group.I work in systems, so I know a little more about it than networking. One thing that I especially like about the systems group is that the professors are extremely nice and very open to discussion: most of the professors have an open door policy, so you can drop in to discuss something at any time. I've heard horror stories from other grad students who meet their advisors once a month - something like that doesn't happen in Wisconsin.Our graduates regularly get jobs at Facebook, Google, Amazon, and pretty much any company you could name. A number of students also become professors: for example, a graduate from our lab, Haryadi Gunawi, became a professor at Chicago; Theophilus Benson became a professor at Duke.If you want to do work in systems or networking, I don't think you could go wrong coming to Wisconsin. We're one of the top groups in both areas.

If you mean the PBS’s / BBC (apparently both got involved) Two Days In October, which part dealt with the protest at University of Wisconsin, it is still up on youtube?Also of note isThis also might be what you are looking for, also called The War At Home and about Wisconsin.The War at Home (1979 film) - Wikipedia“The War at Home is a documentary film about the anti-war movement in the Madison, Wisconsin area during the time of the Vietnam War. It combines archival footage and interviews with participants that explore the events of the period on the University of Wisconsin–Madison campus. The film was nominated for an Academy Award for Best Documentary Feature.[1]”“The film focuses on student protests of government policies in the Vietnam War, clashes between students and police, and the responses of politicians and the public to the turmoil. Among the major events included is the Sterling Hall bombing. Intended to destroy the Army Math Research Center in the building, the bombing caused massive destruction to other parts of the building, resulting in the death of a physics researcher, Robert Fassnacht, who was not involved in the Army Math Research Center.”